High glucose increases nitric oxide generation in lipopolysaccharide-activated macrophages by enhancing activity of protein kinase C-α/δ and NF-κB. (Q39329869)

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English	High glucose increases nitric oxide generation in lipopolysaccharide-activated macrophages by enhancing activity of protein kinase C-α/δ and NF-κB.	scientific article

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scholarly article

1 reference

Europe PubMed Central

11 September 2017

High glucose increases nitric oxide generation in lipopolysaccharide-activated macrophages by enhancing activity of protein kinase C-α/δ and NF-κB. (English)

1 reference

Europe PubMed Central

11 September 2017

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lipopolysaccharide

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author name string

Kuo-Feng Hua

1

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Europe PubMed Central

11 September 2017

Szu-Hsuan Wang

2

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Europe PubMed Central

11 September 2017

Wei-Chih Dong

3

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Europe PubMed Central

11 September 2017

Chai-Yi Lin

4

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Europe PubMed Central

11 September 2017

Chen-Lung Ho

5

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Europe PubMed Central

11 September 2017

Tzu-Hua Wu

6

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Europe PubMed Central

11 September 2017

publication date

16 June 2012

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Europe PubMed Central

11 September 2017

Inflammation Research

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Europe PubMed Central

11 September 2017

61

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Europe PubMed Central

11 September 2017

10

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Europe PubMed Central

11 September 2017

1107-1116

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Europe PubMed Central

11 September 2017

https://scigraph.springernature.com/pub.10.1007/s00011-012-0503-1

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Inflammation and metabolic disorders

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https://api.crossref.org/works/10.1007%2FS00011-012-0503-1

21 January 2018

Impaired glucose tolerance is associated with increased serum concentrations of interleukin 6 and co-regulated acute-phase proteins but not TNF-alpha or its receptors

1 reference

https://api.crossref.org/works/10.1007%2FS00011-012-0503-1

21 January 2018

Plasma interleukin-6, tumour necrosis factor alpha and blood cytokine production in type 2 diabetes

1 reference

https://api.crossref.org/works/10.1007%2FS00011-012-0503-1

21 January 2018

Role of EDRF (nitric oxide) in diabetic renal hyperfiltration.

1 reference

https://api.crossref.org/works/10.1007%2FS00011-012-0503-1

21 January 2018

High glucose induces toll-like receptor expression in human monocytes: mechanism of activation

1 reference

https://api.crossref.org/works/10.1007%2FS00011-012-0503-1

21 January 2018

High glucose induces IL-1beta expression in human monocytes: mechanistic insights

1 reference

https://api.crossref.org/works/10.1007%2FS00011-012-0503-1

21 January 2018

Molecular mechanisms of high glucose-induced cyclooxygenase-2 expression in monocytes

1 reference

https://api.crossref.org/works/10.1007%2FS00011-012-0503-1

21 January 2018

High Glucose-Induced Expression of Proinflammatory Cytokine and Chemokine Genes in Monocytic Cells

1 reference

https://api.crossref.org/works/10.1007%2FS00011-012-0503-1

21 January 2018

Molecular mechanisms of tumor necrosis factor alpha gene expression in monocytic cells via hyperglycemia-induced oxidant stress-dependent and -independent pathways

1 reference

https://api.crossref.org/works/10.1007%2FS00011-012-0503-1

21 January 2018

High glucose increases RAW 264.7 macrophages activation by lipoteichoic acid from Staphylococcus aureus.

1 reference

https://api.crossref.org/works/10.1007%2FS00011-012-0503-1

21 January 2018

Upregulation of cyclooxygenase-II gene and PGE2 production of peritoneal macrophages in diabetic rats

1 reference

https://api.crossref.org/works/10.1007%2FS00011-012-0503-1

21 January 2018

Hyperglycemic levels of glucose inhibit interleukin 1 release from RAW 264.7 murine macrophages by activation of protein kinase C.

1 reference

https://api.crossref.org/works/10.1007%2FS00011-012-0503-1

21 January 2018

High glucose up-regulates lipopolysaccharide-stimulated inflammatory cytokine production via c-jun N-terminal kinase in the monocytic cell line THP-1.

1 reference

https://api.crossref.org/works/10.1007%2FS00011-012-0503-1

21 January 2018

Augmented lipopolysaccharide-induced TNF-alpha production by peritoneal macrophages in type 2 diabetic mice is dependent on elevated glucose and requires p38 MAPK.

1 reference

https://api.crossref.org/works/10.1007%2FS00011-012-0503-1

21 January 2018

The role of inflammatory cytokines in diabetes and its complications

1 reference

https://api.crossref.org/works/10.1007%2FS00011-012-0503-1

21 January 2018

Osthole regulates inflammatory mediator expression through modulating NF-κB, mitogen-activated protein kinases, protein kinase C, and reactive oxygen species

1 reference

https://api.crossref.org/works/10.1007%2FS00011-012-0503-1

21 January 2018

Cytosolic NADH/NAD+, free radicals, and vascular dysfunction in early diabetes mellitus

1 reference

https://api.crossref.org/works/10.1007%2FS00011-012-0503-1

21 January 2018

Glucose or diabetes activates p38 mitogen-activated protein kinase via different pathways

1 reference

https://api.crossref.org/works/10.1007%2FS00011-012-0503-1

21 January 2018

Enhanced expression of inducible nitric oxide synthase in murine macrophages and glomerular mesangial cells by elevated glucose levels: possible mediation via protein kinase C.

1 reference

https://api.crossref.org/works/10.1007%2FS00011-012-0503-1

21 January 2018

Role of interleukin-1beta in the pathogenesis of diabetic retinopathy

1 reference

https://api.crossref.org/works/10.1007%2FS00011-012-0503-1

21 January 2018

Inflammatory cytokines and the risk to develop type 2 diabetes: results of the prospective population-based European Prospective Investigation into Cancer and Nutrition (EPIC)-Potsdam Study

1 reference

https://api.crossref.org/works/10.1007%2FS00011-012-0503-1

21 January 2018

Macrophages from high-risk HLA-DQB1*0201/*0302 type 1 diabetes mellitus patients are hypersensitive to lipopolysaccharide stimulation.

1 reference

https://api.crossref.org/works/10.1007%2FS00011-012-0503-1

21 January 2018

Elevated glucose and diabetes promote interleukin-12 cytokine gene expression in mouse macrophages

1 reference

https://api.crossref.org/works/10.1007%2FS00011-012-0503-1

21 January 2018

Lipopolysaccharide stimulation of ERK1/2 increases TNF-alpha production via Egr-1.

1 reference

https://api.crossref.org/works/10.1007%2FS00011-012-0503-1

21 January 2018

High glucose enhances lipopolysaccharide-stimulated CD14 expression in U937 mononuclear cells by increasing nuclear factor kappaB and AP-1 activities

1 reference

https://api.crossref.org/works/10.1007%2FS00011-012-0503-1

21 January 2018

Radicicol suppresses expression of inducible nitric-oxide synthase by blocking p38 kinase and nuclear factor-kappaB/Rel in lipopolysaccharide-stimulated macrophages

1 reference

https://pubmed.ncbi.nlm.nih.gov/22706318

12 December 2020

based on heuristic

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Decreased production of nitric oxide by LPS-treated J774 macrophages in high-glucose medium

1 reference

https://pubmed.ncbi.nlm.nih.gov/22706318

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

LTA and LPS mediated activation of protein kinases in the regulation of inflammatory cytokines expression in macrophages

1 reference

https://pubmed.ncbi.nlm.nih.gov/22706318

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Acute glucose overload potentiates nitric oxide production in lipopolysaccharide-stimulated macrophages: the role of purinergic receptor activation

1 reference

https://pubmed.ncbi.nlm.nih.gov/22706318

12 December 2020

based on heuristic

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Abnormal renal hemodynamic response to reduced renal perfusion pressure in diabetic rats: role of NO.

1 reference

https://pubmed.ncbi.nlm.nih.gov/22706318

12 December 2020

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Infectious emergencies in patients with diabetes mellitus.

1 reference

https://pubmed.ncbi.nlm.nih.gov/22706318

12 December 2020

based on heuristic

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High glucose increases nitric oxide synthase expression and superoxide anion generation in human aortic endothelial cells

1 reference

https://pubmed.ncbi.nlm.nih.gov/22706318

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1007/S00011-012-0503-1

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Europe PubMed Central

11 September 2017

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Europe PubMed Central

11 September 2017

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