Malathion/oxon and lead acetate increase gene expression and protein levels of transient receptor potential canonical channel subunits TRPC1 and TRPC4 in rat endothelial cells of the blood-brain barrier. (Q39335615)

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English	Malathion/oxon and lead acetate increase gene expression and protein levels of transient receptor potential canonical channel subunits TRPC1 and TRPC4 in rat endothelial cells of the blood-brain barrier.	scientific article

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3 February 2020

Malathion/oxon and lead acetate increase gene expression and protein levels of transient receptor potential canonical channel subunits TRPC1 and TRPC4 in rat endothelial cells of the blood-brain barrier (English)

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3 February 2020

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blood–brain barrier

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inferred from title

2

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3 February 2020

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Pergentino Balbuena

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3 February 2020

Beverly A Rzigalinski

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3 February 2020

Marion Ehrich

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3 February 2020

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1 June 2012

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3 February 2020

International Journal of Toxicology

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3 February 2020

31

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3 February 2020

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3 February 2020

238-249

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3 February 2020

The blood-brain barrier/neurovascular unit in health and disease

1 reference

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Regulation of endothelial cell barrier function by store-operated calcium entry

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