Sepsis-induced urinary concentration defect is related to nitric oxide-dependent inactivation of TonEBP/NFAT5, which downregulates renal medullary solute transport proteins and aquaporin-2. (Q39345540)
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scientific article published on June 2012
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English | Sepsis-induced urinary concentration defect is related to nitric oxide-dependent inactivation of TonEBP/NFAT5, which downregulates renal medullary solute transport proteins and aquaporin-2. |
scientific article published on June 2012 |
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Sepsis-induced urinary concentration defect is related to nitric oxide-dependent inactivation of TonEBP/NFAT5, which downregulates renal medullary solute transport proteins and aquaporin-2. (English)
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Christoph Küper
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Maria-Luisa Fraek
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Hans-Helge Müller
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Franz-Xaver Beck
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Wolfgang Neuhofer
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1 June 2012
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Identifiers
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