Sepsis-induced urinary concentration defect is related to nitric oxide-dependent inactivation of TonEBP/NFAT5, which downregulates renal medullary solute transport proteins and aquaporin-2. (Q39345540)

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scientific article published on June 2012
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Sepsis-induced urinary concentration defect is related to nitric oxide-dependent inactivation of TonEBP/NFAT5, which downregulates renal medullary solute transport proteins and aquaporin-2.
scientific article published on June 2012

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    Sepsis-induced urinary concentration defect is related to nitric oxide-dependent inactivation of TonEBP/NFAT5, which downregulates renal medullary solute transport proteins and aquaporin-2. (English)
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    Christoph Küper
    Maria-Luisa Fraek
    Hans-Helge Müller
    Franz-Xaver Beck
    Wolfgang Neuhofer
    1 June 2012
    1887-1895

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