Activation of the small G-protein Rac by human rhinovirus attenuates the TLR3/IFN-α axis while promoting CCL2 release in human monocyte-lineage cells (Q39439140)

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English	Activation of the small G-protein Rac by human rhinovirus attenuates the TLR3/IFN-α axis while promoting CCL2 release in human monocyte-lineage cells	scientific article

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scholarly article

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Europe PubMed Central

PubMed publication ID

12 September 2017

Activation of the small G-protein Rac by human rhinovirus attenuates the TLR3/IFN-α axis while promoting CCL2 release in human monocyte-lineage cells (English)

1 reference

Europe PubMed Central

PubMed publication ID

12 September 2017

toll-like receptor

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author name string

Michael T Schreiber

1

1 reference

Europe PubMed Central

PubMed publication ID

12 September 2017

Bryce Schuler

2

1 reference

Europe PubMed Central

PubMed publication ID

12 September 2017

LuYuan Li

3

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Europe PubMed Central

PubMed publication ID

12 September 2017

David J Hall

4

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Europe PubMed Central

PubMed publication ID

12 September 2017

publication date

11 October 2012

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Europe PubMed Central

PubMed publication ID

12 September 2017

Innate Immunity

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Europe PubMed Central

PubMed publication ID

12 September 2017

19

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Europe PubMed Central

PubMed publication ID

12 September 2017

3

1 reference

Europe PubMed Central

PubMed publication ID

12 September 2017

278-289

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Europe PubMed Central

PubMed publication ID

12 September 2017

Rhinoviruses, allergic inflammation, and asthma

1 reference

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Comparison of rhinovirus A infection in human primary epithelial and HeLa cells

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Rhinovirus replication in human macrophages induces NF-kappaB-dependent tumor necrosis factor alpha production

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Rhinovirus infection of allergen-sensitized and -challenged mice induces eotaxin release from functionally polarized macrophages.

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The role of p38 MAPK in rhinovirus-induced monocyte chemoattractant protein-1 production by monocytic-lineage cells

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The alveolar macrophage: the forgotten cell in asthma

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Rhinovirus infection induces expression of its own receptor intercellular adhesion molecule 1 (ICAM-1) via increased NF-kappaB-mediated transcription.

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Rhinovirus induces MUC5AC in a human infection model and in vitro via NF-κB and EGFR pathways.

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Role of NF-kappa B in cytokine production induced from human airway epithelial cells by rhinovirus infection

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Human monocytic cells direct the robust release of CXCL10 by bronchial epithelial cells during rhinovirus infection

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Rhinoviruses induce interleukin-8 mRNA and protein production in human monocytes

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Human major group rhinoviruses downmodulate the accessory function of monocytes by inducing IL-10.

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Rhinovirus exposure impairs immune responses to bacterial products in human alveolar macrophages

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Sequencing and analyses of all known human rhinovirus genomes reveal structure and evolution

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Clinical and molecular features of human rhinovirus C.

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Review: rhinoviruses and their ICAM receptors

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A cell adhesion molecule, ICAM-1, is the major surface receptor for rhinoviruses

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Rac1 and PAK1 are upstream of IKK-epsilon and TBK-1 in the viral activation of interferon regulatory factor-3.

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Rhinoviral Infections Activate p38MAP-Kinases Via Membrane Rafts and RhoA

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Serine/threonine kinase PKR: a sentinel kinase that discriminates a signaling pathway mediated by TLR4 from those mediated by TLR3 and TLR9.

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Viral stimuli trigger exaggerated thymic stromal lymphopoietin expression by chronic obstructive pulmonary disease epithelium: role of endosomal TLR3 and cytosolic RIG-I-like helicases

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Protein kinase R contributes to immunity against specific viruses by regulating interferon mRNA integrity

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Antiviral signaling through pattern recognition receptors

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Differential role of TLR- and RLR-signaling in the immune responses to influenza A virus infection and vaccination

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Differential roles of MDA5 and RIG-I helicases in the recognition of RNA viruses

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MDA5 and TLR3 initiate pro-inflammatory signaling pathways leading to rhinovirus-induced airways inflammation and hyperresponsiveness

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Cigarette smoke extract enhances rhinovirus-induced toll-like receptor 3 expression and interleukin-8 secretion in A549 cells

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Role of double-stranded RNA pattern recognition receptors in rhinovirus-induced airway epithelial cell responses

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Human rhinovirus recognition in non-immune cells is mediated by Toll-like receptors and MDA-5, which trigger a synergetic pro-inflammatory immune response

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A TRIF-independent branch of TLR3 signaling

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Absence of MyD88 results in enhanced TLR3-dependent phosphorylation of IRF3 and increased IFN-β and RANTES production

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Toll-like receptor 3 is induced by and mediates antiviral activity against rhinovirus infection of human bronchial epithelial cells

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Co-ordinated role of TLR3, RIG-I and MDA5 in the innate response to rhinovirus in bronchial epithelium

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Quantifying the activation of the small molecular weight G-protein ras.

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Rational design and characterization of a Rac GTPase-specific small molecule inhibitor

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Rhinovirus replication causes RANTES production in primary bronchial epithelial cells

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Identifiers

10.1177/1753425912460709

1 reference

Europe PubMed Central

PubMed publication ID

12 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

12 September 2017

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