Downregulation of VEGF-C expression in lung and colon cancer cells decelerates tumor growth and inhibits metastasis via multiple mechanisms (Q39497610)

12 September 2017

title

Downregulation of VEGF-C expression in lung and colon cancer cells decelerates tumor growth and inhibits metastasis via multiple mechanisms (English)

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12 September 2017

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N Khromova

1

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12 September 2017

P Kopnin

series ordinal

2

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12 September 2017

V Rybko

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3

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12 September 2017

B P Kopnin

series ordinal

4

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12 September 2017

language of work or name

English

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publication date

1 August 2011

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12 September 2017

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12 September 2017

volume

31

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12 September 2017

issue

11

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12 September 2017

page(s)

1389-1397

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Clinical significance of vascular endothelial growth factor C and vascular endothelial growth factor receptor 3 in patients with nonsmall cell lung carcinoma

12 September 2017

cites work

1 reference

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Vascular endothelial growth factor-C promotes the growth and invasion of gallbladder cancer via an autocrine mechanism

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Lymphangiogenesis induced by VEGF-C and VEGF-D promotes metastasis and a poor outcome in breast carcinoma: a retrospective study of 61 cases

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Cancer stem cells: mirage or reality?

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Suppression of tumor lymphangiogenesis and lymph node metastasis by blocking vascular endothelial growth factor receptor 3 signaling.

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Imaging steps of lymphatic metastasis reveals that vascular endothelial growth factor-C increases metastasis by increasing delivery of cancer cells to lymph nodes: therapeutic implications

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Epithelial--mesenchymal and mesenchymal--epithelial transitions in carcinoma progression.

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Proteolytic processing regulates receptor specificity and activity of VEGF-C.

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A model for gene therapy of human hereditary lymphedema

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Functional interaction of VEGF-C and VEGF-D with neuropilin receptors

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p53 hot-spot mutants increase tumor vascularization via ROS-mediated activation of the HIF1/VEGF-A pathway

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Vascular endothelial growth factor C stimulates progression of human gastric cancer via both autocrine and paracrine mechanisms.

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Vascular endothelial growth factor-C-mediated lymphangiogenesis promotes tumour metastasis

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The epithelial-mesenchymal transition generates cells with properties of stem cells

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Signal transduction by VEGF receptors in regulation of angiogenesis and lymphangiogenesis

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EMT, cancer stem cells and drug resistance: an emerging axis of evil in the war on cancer

21 January 2018

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Vascular Endothelial Growth Factor-C (VEGF-C) and its Receptors KDR and flt-4 are Expressed in AIDS-Associated Kaposi’s Sarcoma

21 January 2018

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Vascular endothelial growth factor is an autocrine growth factor in human malignant mesothelioma

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The VEGF-C/Flt-4 axis promotes invasion and metastasis of cancer cells

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Further evidence for expression and function of the VEGF-C/VEGFR-3 axis in cancer cells.

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Lymphangiogenesis: Molecular mechanisms and future promise

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Epithelial-mesenchymal transitions in development and disease

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Migration-promoting role of VEGF-C and VEGF-C binding receptors in human breast cancer cells.

21 January 2018

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Side population cells in human cancers

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Neuropilin-2 mediates VEGF-C-induced lymphatic sprouting together with VEGFR3.

21 January 2018

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Solitary lung tumors and their spontaneous metastasis in athymic nude mice orthotopically implanted with human non-small cell lung cancer

21 January 2018

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Biomarkers for epithelial-mesenchymal transitions

21 January 2018

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A novel vascular endothelial growth factor, VEGF-C, is a ligand for the Flt4 (VEGFR-3) and KDR (VEGFR-2) receptor tyrosine kinases

21 January 2018

1 reference

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Clinical significance of plasma level of vascular endothelial growth factor-C in patients with colorectal cancer

31 March 2020

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Vascular endothelial growth factor-D activates VEGFR-3 expressed in osteoblasts inducing their differentiation

31 March 2020

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Association between lymphangiogenesis-/micrometastasis- and adhesion-related molecules in resected stage I NSCLC

31 March 2020

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31 March 2020

Identifiers

DOI

10.1038/ONC.2011.330

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12 September 2017

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