Receptor for advanced glycation end-products signals through Ras during tobacco smoke-induced pulmonary inflammation. (Q39623825)

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17 January 2020

title

Receptor for advanced glycation end-products signals through Ras during tobacco smoke-induced pulmonary inflammation (English)

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17 January 2020

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1

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17 January 2020

author name string

Stephen D Kasteler

series ordinal

2

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17 January 2020

Robert E Schmitt

series ordinal

3

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17 January 2020

John R Hoidal

series ordinal

4

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17 January 2020

publication date

3 December 2010

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American Journal of Respiratory Cell and Molecular Biology

17 January 2020

published in

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17 January 2020

volume

45

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17 January 2020

issue

2

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17 January 2020

page(s)

411-418

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RAGE: a new target for the prevention and treatment of the vascular and inflammatory complications of diabetes

17 January 2020

cites work

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RAGE mediates a novel proinflammatory axis: a central cell surface receptor for S100/calgranulin polypeptides

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RAGE: developmental expression and positive feedback regulation by Egr-1 during cigarette smoke exposure in pulmonary epithelial cells.

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A new rat type I-like alveolar epithelial cell line R3/1: bleomycin effects on caveolin expression

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The development of emphysema in cigarette smoke-exposed mice is strain dependent

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Midkine is regulated by hypoxia and causes pulmonary vascular remodeling.

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Activation of the receptor for advanced glycation end products triggers a p21(ras)-dependent mitogen-activated protein kinase pathway regulated by oxidant stress.

21 January 2018

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Receptor for advanced glycation end products (RAGE)-mediated neurite outgrowth and activation of NF-kappaB require the cytoplasmic domain of the receptor but different downstream signaling pathways

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Down-regulation of the receptor for advanced glycation end-products (RAGE) supports non-small cell lung carcinoma

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RAGE signaling sustains inflammation and promotes tumor development

21 January 2018

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RAGE (Receptor for Advanced Glycation Endproducts), RAGE ligands, and their role in cancer and inflammation.

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N -Glycans on the receptor for advanced glycation end products influence amphoterin binding and neurite outgrowth

21 January 2018

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Nuclear factor-kappaB: the enemy within

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Nuclear factor-kappaB is constitutively activated in prostate cancer in vitro and is overexpressed in prostatic intraepithelial neoplasia and adenocarcinoma of the prostate.

21 January 2018

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21 January 2018

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Inhibition of NF-kappaB in cancer cells converts inflammation- induced tumor growth mediated by TNFalpha to TRAIL-mediated tumor regression

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Regulation of neutrophil functions by proinflammatory cytokines

21 January 2018

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Inflammatory cells and chronic obstructive pulmonary disease

21 January 2018

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Cigarette smoke combined with Toll-like receptor 3 signaling triggers exaggerated epithelial regulated upon activation, normal T-cell expressed and secreted/CCL5 expression in chronic rhinosinusitis.

21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

Identifiers

DOI

10.1165/RCMB.2010-0231OC

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:21131443%20AND%20SRC:MED&resulttype=core&format=json

17 January 2020

1 reference