Suppression of prostaglandin E2-induced MUC5AC overproduction by RGS4 in the airway (Q39886101)

15 September 2017

title

Suppression of prostaglandin E2-induced MUC5AC overproduction by RGS4 in the airway (English)

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15 September 2017

6

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15 September 2017

Kyoung Seob Song

1

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15 September 2017

Yeon Ho Choi

2

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15 September 2017

Jong-Mu Kim

3

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15 September 2017

Hyunjae Lee

4

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15 September 2017

Tae-Jin Lee

5

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15 September 2017

publication date

6 February 2009

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American Journal of Physiology - Lung Cellular and Molecular Physiology

15 September 2017

published in

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volume

296

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15 September 2017

issue

4

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15 September 2017

page(s)

L684-92

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Prostaglandin F(2alpha) receptor in the corpus luteum: recent information on the gene, messenger ribonucleic acid, and protein

15 September 2017

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Prostaglandin E2 induces MUC8 gene expression via a mechanism involving ERK MAPK/RSK1/cAMP response element binding protein activation in human airway epithelial cells

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Prostaglandin E2 receptors of the EP2 and EP4 subtypes regulate activation and differentiation of mouse B lymphocytes to IgE-secreting cells

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Phosphorylation of glycogen synthase kinase-3 and stimulation of T-cell factor signaling following activation of EP2 and EP4 prostanoid receptors by prostaglandin E2.

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Interleukin-1beta-induced mucin production in human airway epithelium is mediated by cyclooxygenase-2, prostaglandin E2 receptors, and cyclic AMP-protein kinase A signaling.

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

Identifiers

DOI

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15 September 2017

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