c-Jun N-terminal kinase activation failure is a new mechanism of anthracycline resistance in acute myeloid leukemia (Q39957937)

15 September 2017

title

c-Jun N-terminal kinase activation failure is a new mechanism of anthracycline resistance in acute myeloid leukemia (English)

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15 September 2017

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E D Lagadinou

1

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15 September 2017

P G Ziros

2

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15 September 2017

O A Tsopra

3

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15 September 2017

K Dimas

4

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D Kokkinou

5

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E Thanopoulou

6

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M Karakantza

7

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P Pantazis

8

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A Spyridonidis

9

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N C Zoumbos

10

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15 September 2017

publication date

24 July 2008

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15 September 2017

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15 September 2017

volume

22

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15 September 2017

issue

10

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15 September 2017

page(s)

1899-1908

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https://scigraph.springernature.com/pub.10.1038/leu.2008.192

15 September 2017

exact match

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cites work

Acute myeloid leukaemia

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Mitoxantrone, etoposide, and cytarabine with or without valspodar in patients with relapsed or refractory acute myeloid leukemia and high-risk myelodysplastic syndrome: a phase III trial (E2995)

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Addition of cyclosporin-A to chemotherapy in secondary (post-MDS) AML in the elderly. A multicenter randomized trial of the Leukemia Working Group of the Hellenic Society of Hematology.

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Receptor- and mitochondrial-mediated apoptosis in acute leukemia: a translational view

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7 January 2021

High bcl-2 expression in acute myeloid leukemia cells correlates with CD34 positivity and complete remission rate.

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7 January 2021

Signal transduction by the JNK group of MAP kinases

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7 January 2021

Role of mitogen-activated protein kinases in the response of tumor cells to chemotherapy

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Cellular stress response and apoptosis in cancer therapy.

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7 January 2021

Role for c-jun N-terminal kinase in treatment-refractory acute myeloid leukemia (AML): signaling to multidrug-efflux and hyperproliferation

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7 January 2021

ERK1/2 phosphorylation is an independent predictor of complete remission in newly diagnosed adult acute lymphoblastic leukemia

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Deeper insights into hematological oncology disorders via single-cell phospho-signaling analysis

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Implication of radical oxygen species in ceramide generation, c-Jun N-terminal kinase activation and apoptosis induced by daunorubicin

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Signaling pathways activated by daunorubicin

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7 January 2021

Long-term activation of SAPK/JNK, p38 kinase and fas-L expression by cisplatin is attenuated in human carcinoma cells that acquired drug resistance.

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7 January 2021

Essential role of c-Jun-NH2-terminal kinase on synergy induction of apoptosis by TRAIL plus ADM in ADM resistant MCF-7/ADM cells

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7 January 2021

Downregulation of JNK/SAPK activity is associated with the cross-resistance to P-glycoprotein-unrelated drugs in multidrug-resistant FM3A/M cells overexpressing P-glycoprotein

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7 January 2021

Reversing chemoresistance in cisplatin-resistant human ovarian cancer cells: a role of c-Jun NH2-terminal kinase 1.

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The bone-specific transcriptional regulator Cbfa1 is a target of mechanical signals in osteoblastic cells.

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Transformation of mammalian cells by constitutively active MAP kinase kinase

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7 January 2021

MDR1 gene overexpression confers resistance to imatinib mesylate in leukemia cell line models

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7 January 2021

Requirement of JNK for stress-induced activation of the cytochrome c-mediated death pathway

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7 January 2021

Reversal of P-glycoprotein-mediated multidrug resistance in cancer cells by the c-Jun NH2-terminal kinase

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7 January 2021

Methylation-controlled J protein promotes c-Jun degradation to prevent ABCB1 transporter expression

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7 January 2021

The tyrphostin adaphostin interacts synergistically with proteasome inhibitors to induce apoptosis in human leukemia cells through a reactive oxygen species (ROS)-dependent mechanism

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https://api.crossref.org/works/10.1038%2FLEU.2008.192

7 January 2021

Farnesyltransferase inhibitors interact synergistically with the Chk1 inhibitor UCN-01 to induce apoptosis in human leukemia cells through interruption of both Akt and MEK/ERK pathways and activation of SEK1/JNK.

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7 January 2021

Synergistic antileukemic interactions between 2-medroxyestradiol (2-ME) and histone deacetylase inhibitors involve Akt down-regulation and oxidative stress

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https://api.crossref.org/works/10.1038%2FLEU.2008.192

7 January 2021

JNK activation is a mediator of arsenic trioxide-induced apoptosis in acute promyelocytic leukemia cells

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https://api.crossref.org/works/10.1038%2FLEU.2008.192

7 January 2021

Pharmacodynamics of cytarabine alone and in combination with 7-hydroxystaurosporine (UCN-01) in AML blasts in vitro and during a clinical trial

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https://api.crossref.org/works/10.1038%2FLEU.2008.192

7 January 2021

Glutathione S-transferase P1-1 (GSTP1-1) inhibits c-Jun N-terminal kinase (JNK1) signaling through interaction with the C terminus

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https://api.crossref.org/works/10.1038%2FLEU.2008.192

7 January 2021

Mitogen-activated protein kinase phosphatase-1 is required for cisplatin resistance

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https://api.crossref.org/works/10.1038%2FLEU.2008.192

7 January 2021

Death versus survival: functional interaction between the apoptotic and stress-inducible heat shock protein pathways

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https://api.crossref.org/works/10.1038%2FLEU.2008.192

7 January 2021

Inhibition of JNK activation through NF-kappaB target genes

1 reference

https://api.crossref.org/works/10.1038%2FLEU.2008.192

7 January 2021

The novel Akt inhibitor, perifosine, induces caspase-dependent apoptosis and downregulates P-glycoprotein expression in multidrug-resistant human T-acute leukemia cells by a JNK-dependent mechanism

1 reference

https://api.crossref.org/works/10.1038%2FLEU.2008.192

7 January 2021

Identifiers

DOI

10.1038/LEU.2008.192

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15 September 2017

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