Aplidin synergizes with cytosine arabinoside: functional relevance of mitochondria in Aplidin-induced cytotoxicity (Q40090768)

17 September 2017

Aplidin synergizes with cytosine arabinoside: functional relevance of mitochondria in Aplidin-induced cytotoxicity (English)

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17 September 2017

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Humeniuk R

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17 September 2017

Menon LG

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17 September 2017

Mishra PJ

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17 September 2017

Saydam G

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Longo-Sorbello GS

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Elisseyeff Y

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Lewis LD

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Aracil M

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Jimeno J

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17 September 2017

Bertino JR

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Banerjee D

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23 August 2007

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2399-2405

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https://scigraph.springernature.com/pub.10.1038/sj.leu.2404911

17 September 2017

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Aplidin induces apoptosis in human cancer cells via glutathione depletion and sustained activation of the epidermal growth factor receptor, Src, JNK, and p38 MAPK.

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JNK activation is critical for Aplidin-induced apoptosis.

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Oxidation and inactivation of low molecular weight protein tyrosine phosphatase by the anticancer drug Aplidin.

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Establishment and characterisation of a human carcinoma cell line with acquired resistance to Aplidin

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In vitro cytotoxicity of aplidin and crossresistance with other cytotoxic drugs in childhood leukemic and normal bone marrow and blood samples: a rational basis for clinical development

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A new melanoma antigen fatty acid-binding protein 7, involved in proliferation and invasion, is a potential target for immunotherapy and molecular target therapy

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Prevention of mitochondrial injury by manganese superoxide dismutase reveals a primary mechanism for alkaline-induced cell death

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Oxidative stress mediates neuronal DNA damage and apoptosis in response to cytosine arabinoside

21 January 2018

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21 January 2018

Calcium and mitochondria

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Aplidin induces the mitochondrial apoptotic pathway via oxidative stress-mediated JNK and p38 activation and protein kinase C delta

21 January 2018

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7 January 2021

Aplidin induces JNK-dependent apoptosis in human breast cancer cells via alteration of glutathione homeostasis, Rac1 GTPase activation, and MKP-1 phosphatase downregulation

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Use of flow and laser-scanning cytometry in analysis of cell death

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Quantitative Analysis of Spontaneous Mitochondrial Depolarizations

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Mitochondria as targets for detection and treatment of cancer.

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7 January 2021

Identifiers

DOI

10.1038/SJ.LEU.2404911

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Dimensions Publication ID

17 September 2017

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