A20/TNFAIP3, a new estrogen-regulated gene that confers tamoxifen resistance in breast cancer cells. (Q40171629)

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25 December 2019

title

A20/TNFAIP3, a new estrogen-regulated gene that confers tamoxifen resistance in breast cancer cells (English)

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25 December 2019

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4

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25 December 2019

author name string

J A Vendrell

1

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25 December 2019

S Ghayad

2

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25 December 2019

S Ben-Larbi

3

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25 December 2019

N Mechti

5

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25 December 2019

P A Cohen

6

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25 December 2019

language of work or name

English

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publication date

12 February 2007

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25 December 2019

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25 December 2019

volume

26

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25 December 2019

issue

32

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25 December 2019

page(s)

4656-4667

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https://scigraph.springernature.com/pub.10.1038/sj.onc.1210269

25 December 2019

exact match

0 references

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Long-term exposure to tamoxifen induces hypersensitivity to estradiol

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Tumor necrosis factor-alpha-induced protein 3 as a putative regulator of nuclear factor-kappaB-mediated resistance to O6-alkylating agents in human glioblastomas

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A20 RNA expression is associated with undifferentiated nasopharyngeal carcinoma and poorly differentiated head and neck squamous cell carcinoma

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Progesterone receptor quantification with radiolabeled promegestone (R 5020) in frozen sections of endometrium and breast cancer tissue

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A20 inhibits cytokine-induced apoptosis and nuclear factor kappaB-dependent gene activation in islets

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Acquired tamoxifen resistance in human breast cancer--potential mechanisms and clinical implications

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Modulation of tamoxifen sensitivity by antisense Bcl-2 and trastuzumab in breast carcinoma cells

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Failure to regulate TNF-induced NF-kappaB and cell death responses in A20-deficient mice

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A20 inhibits oxidized low-density lipoprotein-induced apoptosis through negative Fas/Fas ligand-dependent activation of caspase-8 and mitochondrial pathways in murine RAW264.7 macrophages.

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A20 protects mice from lethal radical hepatectomy by promoting hepatocyte proliferation via a p21waf1-dependent mechanism

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7 January 2021

ACTR/AIB1 functions as an E2F1 coactivator to promote breast cancer cell proliferation and antiestrogen resistance

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Enhanced estrogen receptor (ER) alpha, ERBB2, and MAPK signal transduction pathways operate during the adaptation of MCF-7 cells to long term estrogen deprivation

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ABIN-1 binds to NEMO/IKKgamma and co-operates with A20 in inhibiting NF-kappaB

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Inducible upregulation of oestrogen receptor-beta1 affects oestrogen and tamoxifen responsiveness in MCF7 human breast cancer cells

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Upregulation of PKC-delta contributes to antiestrogen resistance in mammary tumor cells.

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Hepatitis C virus core protein induces expression of genes regulating immune evasion and anti-apoptosis in hepatocytes

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An inhibitory effect of A20 on NF-kappaB activation in airway epithelium upon influenza virus infection

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7 January 2021

A20, a modulator of smooth muscle cell proliferation and apoptosis, prevents and induces regression of neointimal hyperplasia

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Mechanisms of tamoxifen resistance

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Activation of the B-cell surface receptor CD40 induces A20, a novel zinc finger protein that inhibits apoptosis

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Functional dichotomy of A20 in apoptotic and necrotic cell death

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Estrogen regulation in human breast cancer cells of new downstream gene targets involved in estrogen metabolism, cell proliferation and cell transformation

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De-ubiquitination and ubiquitin ligase domains of A20 downregulate NF-kappaB signalling

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Interaction of TAFII105 with selected p65/RelA dimers is associated with activation of subset of NF-kappa B genes

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Activation of the MAPK pathway enhances sensitivity of MCF-7 breast cancer cells to the mitogenic effect of estradiol

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A20 inhibits NF-kappa B activation downstream of multiple Map3 kinases and interacts with the I kappa B signalosome

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Identifiers

DOI

10.1038/SJ.ONC.1210269

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:17297453%20AND%20SRC:MED&resulttype=core&format=json

Dimensions Publication ID

25 December 2019

0 references

1 reference