The murine TRAIL receptor signals caspase-independent cell death through ceramide (Q40222818)

18 September 2017

The murine TRAIL receptor signals caspase-independent cell death through ceramide (English)

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18 September 2017

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Lutz Thon

1

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18 September 2017

Sabine Mathieu

series ordinal

2

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18 September 2017

Dieter Kabelitz

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3

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18 September 2017

Dieter Adam

series ordinal

4

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18 September 2017

language of work or name

English

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publication date

30 August 2006

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Experimental Cell Research

18 September 2017

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18 September 2017

312

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18 September 2017

19

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18 September 2017

3808-3821

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Four deaths and a funeral: from caspases to alternative mechanisms.

18 September 2017

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Apoptotic and necrotic cell death induced by death domain receptors.

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Caspase-8 serves both apoptotic and nonapoptotic roles

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Death receptors and caspases: role in lymphocyte proliferation, cell death, and autoimmunity

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Origin and evolution of eukaryotic apoptosis: the bacterial connection

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Overexpression of acid ceramidase protects from tumor necrosis factor-induced cell death

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Inhibition of caspases increases the sensitivity of L929 cells to necrosis mediated by tumor necrosis factor.

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Ceramide mediates caspase-independent programmed cell death

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Dual signaling of the Fas receptor: initiation of both apoptotic and necrotic cell death pathways

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Necrotic death pathway in Fas receptor signaling

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Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule

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A novel anti-human DR5 monoclonal antibody with tumoricidal activity induces caspase-dependent and caspase-independent cell death

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Ceramide in apoptosis: an overview and current perspectives

7 January 2021

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Regulation of ceramide production and apoptosis

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Biochemical characteristics of caspases-3, -6, -7, and -8

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Death receptor-induced apoptotic and necrotic cell death: differential role of caspases and mitochondria.

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Synthetic peptides corresponding to ligand-binding region of death receptors, DR5, Fas, and TNFR, specifically inhibit cell death mediated by the death ligands, respectively

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Involvement of tumor necrosis factor-related apoptosis-inducing ligand in NK cell-mediated and IFN-gamma-dependent suppression of subcutaneous tumor growth

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Caspase cleavage product lacking amino-terminus of IkappaBalpha sensitizes resistant cells to TNF-alpha and TRAIL-induced apoptosis

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Caspase-independent cell death in T lymphocytes

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Cleavage of poly(ADP-ribose) polymerase by a proteinase with properties like ICE

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Regulation of an ATG7-beclin 1 program of autophagic cell death by caspase-8

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Induction of tumor-specific T cell immunity by anti-DR5 antibody therapy

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Chemical inhibitor of nonapoptotic cell death with therapeutic potential for ischemic brain injury

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Non-specific effects of methyl ketone peptide inhibitors of caspases

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Cathepsin B acts as a dominant execution protease in tumor cell apoptosis induced by tumor necrosis factor

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Multiple pathways of TWEAK-induced cell death

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The ectodermal dysplasia receptor activates the nuclear factor-kappaB, JNK, and cell death pathways and binds to ectodysplasin A

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Mediation of poly(ADP-ribose) polymerase-1-dependent cell death by apoptosis-inducing factor

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Poly(ADP-ribose) polymerase is a mediator of necrotic cell death by ATP depletion

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The therapeutic potential of modulating the ceramide/sphingomyelin pathway

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Stimulation of RNA Synthesis in L-929 Cells by Rabbit Tumor Necrosis Factor

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inferred from DOI database lookup

7 January 2021

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Identifiers

DOI

10.1016/J.YEXCR.2006.08.017

1 reference

18 September 2017

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