Evidence that TNF-TNFR1-TRADD-TRAF2-RIP-TAK1-IKK pathway mediates constitutive NF-kappaB activation and proliferation in human head and neck squamous cell carcinoma (Q40235625)

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scientific article published on 4 September 2006

Language	Label	Description	Also known as
English	Evidence that TNF-TNFR1-TRADD-TRAF2-RIP-TAK1-IKK pathway mediates constitutive NF-kappaB activation and proliferation in human head and neck squamous cell carcinoma	scientific article published on 4 September 2006

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scholarly article

1 reference

Europe PubMed Central

18 September 2017

Evidence that TNF-TNFR1-TRADD-TRAF2-RIP-TAK1-IKK pathway mediates constitutive NF-kappaB activation and proliferation in human head and neck squamous cell carcinoma (English)

1 reference

Europe PubMed Central

18 September 2017

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author name string

Jackson-Bernitsas DG

1

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Europe PubMed Central

18 September 2017

Ichikawa H

2

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Europe PubMed Central

18 September 2017

Takada Y

3

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Europe PubMed Central

18 September 2017

Myers JN

4

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Europe PubMed Central

18 September 2017

Lin XL

5

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Europe PubMed Central

18 September 2017

Darnay BG

6

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Europe PubMed Central

18 September 2017

Chaturvedi MM

7

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Europe PubMed Central

18 September 2017

Aggarwal BB

8

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Europe PubMed Central

18 September 2017

language of work or name

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publication date

4 September 2006

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Europe PubMed Central

18 September 2017

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Europe PubMed Central

18 September 2017

26

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Europe PubMed Central

18 September 2017

10

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18 September 2017

1385-1397

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18 September 2017

https://scigraph.springernature.com/pub.10.1038/sj.onc.1209945

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Signalling pathways of the TNF superfamily: a double-edged sword

1 reference

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Nuclear factor-kappaB: the enemy within

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Inhibition of growth and survival of human head and neck squamous cell carcinoma cells by curcumin via modulation of nuclear factor-kappaB signaling

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TAK1 is recruited to the tumor necrosis factor-alpha (TNF-alpha) receptor 1 complex in a receptor-interacting protein (RIP)-dependent manner and cooperates with MEKK3 leading to NF-kappaB activation

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The nuclear transcription factor kappaB/bcl-2 pathway correlates with pathologic complete response to doxorubicin-based neoadjuvant chemotherapy in human breast cancer.

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TRAF6 is a signal transducer for interleukin-1

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Assay for redox-sensitive transcription factor

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Activation of NF-kappaB by RANK requires tumor necrosis factor receptor-associated factor (TRAF) 6 and NF-kappaB-inducing kinase. Identification of a novel TRAF6 interaction motif

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Activation of IKK by TNFalpha requires site-specific ubiquitination of RIP1 and polyubiquitin binding by NEMO

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Lymphotoxin is an autocrine growth factor for Epstein-Barr virus-infected B cell lines

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Oncogenic Ha-Ras-induced signaling activates NF-kappaB transcriptional activity, which is required for cellular transformation

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Missing pieces in the NF-kappaB puzzle

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Association of activated transcription factor nuclear factor kappab with chemoradiation resistance and poor outcome in esophageal carcinoma

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NF-kappaB regulation in the immune system

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Nuclear factor-kappaB is an important modulator of the altered gene expression profile and malignant phenotype in squamous cell carcinoma.

1 reference

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Curcumin suppresses growth of head and neck squamous cell carcinoma

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Thalidomide suppresses NF-kappa B activation induced by TNF and H2O2, but not that activated by ceramide, lipopolysaccharides, or phorbol ester

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Requirement of NF-kappaB activation to suppress p53-independent apoptosis induced by oncogenic Ras.

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NF-kappaB activation by tumour necrosis factor requires the Akt serine-threonine kinase

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Two pathways to NF-kappaB.

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NF-kappaB is a target of AKT in anti-apoptotic PDGF signalling

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Nuclear factor-kappaB: a friend or a foe in cancer?

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Protein farnesyltransferase inhibitor (SCH 66336) abolishes NF-kappaB activation induced by various carcinogens and inflammatory stimuli leading to suppression of NF-kappaB-regulated gene expression and up-regulation of apoptosis

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Hydrogen peroxide activates NF-kappa B through tyrosine phosphorylation of I kappa B alpha and serine phosphorylation of p65: evidence for the involvement of I kappa B alpha kinase and Syk protein-tyrosine kinase

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Cytokines IL-1α, IL-6, and GM-CSF constitutively secreted by oral squamous carcinoma induce down-regulation of CD80 costimulatory molecule expression: restoration by interferon γ

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Ubiquitination of RIP Is Required for Tumor Necrosis Factor α-induced NF-κB Activation

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Effects of pharmacologic antagonists of epidermal growth factor receptor, PI3K and MEK signal kinases on NF-kappaB and AP-1 activation and IL-8 and VEGF expression in human head and neck squamous cell carcinoma lines

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Constitutive activation of NF-kappaB causes resistance to apoptosis in human cutaneous T cell lymphoma HuT-78 cells. Autocrine role of tumor necrosis factor and reactive oxygen intermediates

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IFN-alpha suppresses activation of nuclear transcription factors NF-kappa B and activator protein 1 and potentiates TNF-induced apoptosis

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Constitutive activation of transcription factors NF-(kappa)B, AP-1, and NF-IL6 in human head and neck squamous cell carcinoma cell lines that express pro-inflammatory and pro-angiogenic cytokines

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Activation of transcription factor NF-kappa B is suppressed by curcumin (diferuloylmethane) [corrected]

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Clinical significance of interleukin-6 and interleukin-6 receptor expressions in oral squamous cell carcinoma

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10.1038/SJ.ONC.1209945

1 reference

Europe PubMed Central

18 September 2017

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1 reference

Europe PubMed Central

18 September 2017

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