Methods for studying the cellular response to DNA damage: influence of the Mre11 complex on chromosome metabolism. (Q40263773)

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25 December 2019

title

Methods for studying the cellular response to DNA damage: influence of the Mre11 complex on chromosome metabolism (English)

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25 December 2019

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2

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25 December 2019

author name string

Jan-Willem F Theunissen

series ordinal

1

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25 December 2019

language of work or name

English

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publication date

1 January 2006

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25 December 2019

published in

Methods in Enzymology

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25 December 2019

volume

409

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25 December 2019

page(s)

251-284

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Initiating cellular stress responses

25 December 2019

cites work

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Methods for the quantification of DNA double-strand breaks determined from the distribution of DNA fragment sizes measured by pulsed-field gel electrophoresis

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Wild-type p53 is a cell cycle checkpoint determinant following irradiation

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hMre11 and hRad50 nuclear foci are induced during the normal cellular response to DNA double-strand breaks

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7 January 2021

DNA damage-dependent nuclear dynamics of the Mre11 complex

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Suppression of spontaneous chromosomal rearrangements by S phase checkpoint functions in Saccharomyces cerevisiae

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Radioresistant DNA synthesis: an intrinsic feature of ataxia telangiectasia

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The cellular response to DNA double-strand breaks: defining the sensors and mediators

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Mitotic recombination produces the majority of recessive fibroblast variants in heterozygous mice.

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The Mre11 complex and the metabolism of chromosome breaks: the importance of communicating and holding things together

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7 January 2021

Checkpoint failure and chromosomal instability without lymphomagenesis in Mre11(ATLD1/ATLD1) mice

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Two molecularly distinct G(2)/M checkpoints are induced by ionizing irradiation

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Dual roles of ATM in the cellular response to radiation and in cell growth control.

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Cell death, chromosome damage and mitotic delay in normal human, ataxia telangiectasia and retinoblastoma fibroblasts after x-irradiation

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Identifiers

DOI

1 reference

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25 December 2019

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