Carboxyl-terminal fragment of amyloid precursor protein and hydrogen peroxide induce neuronal cell death through different pathways. (Q40271630)

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29 January 2020

title

Carboxyl-terminal fragment of amyloid precursor protein and hydrogen peroxide induce neuronal cell death through different pathways (English)

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29 January 2020

3

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29 January 2020

Eduard Rodríguez-Farré

6

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29 January 2020

Coral Sanfeliu

8

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29 January 2020

author name string

J Sebastià

1

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29 January 2020

M Pertusa

2

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29 January 2020

A M Planas

4

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29 January 2020

R Verbeek

5

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29 January 2020

R Cristòfol

7

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29 January 2020

publication date

6 June 2006

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Journal of Neural Transmission

29 January 2020

published in

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29 January 2020

volume

113

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29 January 2020

issue

12

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29 January 2020

page(s)

1837-1845

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Amyloid beta peptide-induced cerebral neuronal loss is mediated by caspase-3 in vivo

29 January 2020

cites work

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Crossref

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Reduced risk of Alzheimer disease in users of antioxidant vitamin supplements: the Cache County Study

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Vitamin E protects neurons against oxidative cell death in vitro more effectively than 17-beta estradiol and induces the activity of the transcription factor NF-kappaB

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Chronological primacy of oxidative stress in Alzheimer disease.

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Processing of the Amyloid Protein Precursor to Potentially Amyloidogenic Derivatives

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Roles of A beta and carboxyl terminal peptide fragments of amyloid precursor protein in Alzheimer disease

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Multiple signaling events in amyloid beta-induced, oxidative stress-dependent neuronal apoptosis

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An etiological role of amyloidogenic carboxyl-terminal fragments of the beta-amyloid precursor protein in Alzheimer's disease.

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The amyloid hypothesis of Alzheimer's disease: progress and problems on the road to therapeutics

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Oxidative stress mechanisms and potential therapeutics in Alzheimer disease

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Deciphering the molecular basis of memory failure in Alzheimer's disease

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Measurement of striatal H2O2 by microdialysis following global forebrain ischemia and reperfusion in the rat: correlation with the cytotoxic potential of H2O2 in vitro

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Cytotoxicity mediated by conditional expression of a carboxyl-terminal derivative of the beta-amyloid precursor protein

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Re-examination and further development of a precise and rapid dye method for measuring cell growth/cell kill

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Cellular response to oxidative stress: signaling for suicide and survival

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A role for 4-hydroxynonenal, an aldehydic product of lipid peroxidation, in disruption of ion homeostasis and neuronal death induced by amyloid beta-peptide

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Synaptic targeting by Alzheimer's-related amyloid beta oligomers.

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Alpha-lipoic acid protects rat cortical neurons against cell death induced by amyloid and hydrogen peroxide through the Akt signalling pathway

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Hydrogen peroxide mediates amyloid beta protein toxicity

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Pathophysiological roles of amyloidogenic carboxy-terminal fragments of the beta-amyloid precursor protein in Alzheimer's disease

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Heme oxygenase expression in human central nervous system disorders

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Beta-amyloid precursor protein of Alzheimer disease occurs as 110- to 135-kilodalton membrane-associated proteins in neural and nonneural tissues.

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Is oxidative damage the fundamental pathogenic mechanism of Alzheimer's and other neurodegenerative diseases?

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Lipid peroxidation and oxidative imbalance: early functional events in Alzheimer's disease.

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Neurodegenerative mechanisms in Alzheimer disease. A role for oxidative damage in amyloid beta protein precursor-mediated cell death

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Protective role of heme oxygenase-1 in oxidative stress-induced neuronal injury

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Amyloid beta-induced changes in nitric oxide production and mitochondrial activity lead to apoptosis

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12 December 2020

Oxidative stress potentiates BACE1 gene expression and Abeta generation

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12 December 2020

Amyloid &#946;-Peptide [1-42]-Associated Free Radical-Induced Oxidative Stress And Neurodegeneration in Alzheimers Disease Brain: Mechanisms and Consequences

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12 December 2020

Early vitamin E supplementation in young but not aged mice reduces Abeta levels and amyloid deposition in a transgenic model of Alzheimer's disease

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12 December 2020

Effects of carboxyl-terminal fragment of Alzheimer's amyloid precursor protein and amyloid beta-peptide on the production of cytokines and nitric oxide in glial cells

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12 December 2020

Neuronal expression of beta-amyloid precursor protein Alzheimer mutations causes intracellular accumulation of a C-terminal fragment containing both the amyloid beta and cytoplasmic domains

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12 December 2020

Novel tricyclic pyrone compounds prevent intracellular APP C99-induced cell death

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12 December 2020

Redox-active iron mediates amyloid-beta toxicity

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12 December 2020

Apolipoprotein E isoforms and apolipoprotein AI protect from amyloid precursor protein carboxy terminal fragment-associated cytotoxicity

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12 December 2020

Nordihydroguaiaretic acid protects hippocampal neurons against amyloid beta-peptide toxicity, and attenuates free radical and calcium accumulation

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12 December 2020

Trolox and 17beta-estradiol protect against amyloid beta-peptide neurotoxicity by a mechanism that involves modulation of the Wnt signaling pathway

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12 December 2020

Beta-amyloid fragment 25-35 causes mitochondrial dysfunction in primary cortical neurons

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12 December 2020

10.1007/S00702-006-0492-8

Identifiers

DOI

1 reference

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Dimensions Publication ID

29 January 2020

1034038030

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