Poly(ADP-ribose) polymerase-1 signaling to mitochondria in necrotic cell death requires RIP1/TRAF2-mediated JNK1 activation (Q40323235)

19 September 2017

Poly(ADP-ribose) polymerase-1 signaling to mitochondria in necrotic cell death requires RIP1/TRAF2-mediated JNK1 activation (English)

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19 September 2017

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Yue Xu

1

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19 September 2017

Shuang Huang

series ordinal

2

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19 September 2017

Zheng-Gang Liu

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3

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19 September 2017

Jiahuai Han

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4

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19 September 2017

language of work or name

English

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publication date

30 January 2006

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Journal of Biological Chemistry

19 September 2017

8

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based on heuristic

inferred from page(s)

published in

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19 September 2017

281

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19 September 2017

13

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19 September 2017

8788-8795

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Poly(ADP-ribosyl)ation reactions in the regulation of nuclear functions

19 September 2017

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https://api.crossref.org/works/10.1074%2FJBC.M508135200

Structure and function of poly(ADP-ribose) polymerase

21 January 2018

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https://api.crossref.org/works/10.1074%2FJBC.M508135200

Poly(ADP-ribose) polymerase is a mediator of necrotic cell death by ATP depletion

21 January 2018

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https://api.crossref.org/works/10.1074%2FJBC.M508135200

Failure of poly(ADP-ribose) polymerase cleavage by caspases leads to induction of necrosis and enhanced apoptosis

21 January 2018

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https://api.crossref.org/works/10.1074%2FJBC.M508135200

Poly(ADP-ribose) polymerase gene disruption renders mice resistant to cerebral ischemia.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

Ischemic brain injury is mediated by the activation of poly(ADP-ribose)polymerase.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

Mice lacking the poly(ADP-ribose) polymerase gene are resistant to pancreatic beta-cell destruction and diabetes development induced by streptozocin.

21 January 2018

1 reference

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The therapeutic potential of poly(ADP-ribose) polymerase inhibitors

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

Nitric oxide activation of poly(ADP-ribose) synthetase in neurotoxicity

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

Alkylating DNA damage stimulates a regulated form of necrotic cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

Mediation of poly(ADP-ribose) polymerase-1-dependent cell death by apoptosis-inducing factor

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

Poly(ADP-ribose) polymerase-1-mediated cell death in astrocytes requires NAD+ depletion and mitochondrial permeability transition

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

JNK potentiates TNF-stimulated necrosis by increasing the production of cytotoxic reactive oxygen species

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

Reactive nitrogen species-induced cell death requires Fas-dependent activation of c-Jun N-terminal kinase

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

Reactive oxygen species promote TNFalpha-induced death and sustained JNK activation by inhibiting MAP kinase phosphatases

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

Signal transduction by tumor necrosis factor and its relatives

21 January 2018

1 reference

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TNF-R1 signaling: a beautiful pathway

21 January 2018

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https://api.crossref.org/works/10.1074%2FJBC.M508135200

Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

Tumor necrosis factor-induced nonapoptotic cell death requires receptor-interacting protein-mediated cellular reactive oxygen species accumulation

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

The stress-activated protein kinase pathways

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

Human hepatitis B virus-X protein alters mitochondrial function and physiology in human liver cells

21 January 2018

1 reference

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Measurement of mitochondrial membrane potential using fluorescent rhodamine derivatives

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

The permeability transition pore. Control points of a cyclosporin A-sensitive mitochondrial channel involved in cell death

21 January 2018

1 reference

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A new method for the cytofluorimetric analysis of mitochondrial membrane potential using the J-aggregate forming lipophilic cation 5,5',6,6'-tetrachloro-1,1',3,3'-tetraethylbenzimidazolcarbocyanine iodide (JC-1).

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

Molecular characterization of mitochondrial apoptosis-inducing factor

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

RIP kinase is involved in arsenic-induced apoptosis in multiple myeloma cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

Essential roles of receptor-interacting protein and TRAF2 in oxidative stress-induced cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

The death domain kinase RIP1 is essential for tumor necrosis factor alpha signaling to p38 mitogen-activated protein kinase

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

The role of the death-domain kinase RIP in tumour-necrosis-factor-induced activation of mitogen-activated protein kinases

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

ASK1 is required for sustained activations of JNK/p38 MAP kinases and apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

Requirement of JNK for stress-induced activation of the cytochrome c-mediated death pathway

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

P38 alpha mitogen-activated protein kinase sensitizes cells to apoptosis induced by different stimuli.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

A peptide inhibitor of c-Jun N-terminal kinase protects against excitotoxicity and cerebral ischemia

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

D-JNKI1, a cell-penetrating c-Jun-N-terminal kinase inhibitor, protects against cell death in severe cerebral ischemia

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

DNA damaging agents induce expression of Fas ligand and subsequent apoptosis in T lymphocytes via the activation of NF-kappa B and AP-1.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

Dominant-negative c-Jun promotes neuronal survival by reducing BIM expression and inhibiting mitochondrial cytochrome c release

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

Execution of Apoptosis Signal-regulating Kinase 1 (ASK1)-induced Apoptosis by the Mitochondria-dependent Caspase Activation

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

Bcl-2 undergoes phosphorylation by c-Jun N-terminal kinase/stress-activated protein kinases in the presence of the constitutively active GTP-binding protein Rac1.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

BCL-2 is phosphorylated and inactivated by an ASK1/Jun N-terminal protein kinase pathway normally activated at G(2)/M.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

Bcl-2 phosphorylation required for anti-apoptosis function

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

Dissection of TNF receptor 1 effector functions: JNK activation is not linked to apoptosis while NF-kappaB activation prevents cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

Caspase-independent cell death by arsenic trioxide in human cervical cancer cells: reactive oxygen species-mediated poly(ADP-ribose) polymerase-1 activation signals apoptosis-inducing factor release from mitochondria.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

Activation and caspase-mediated inhibition of PARP: a molecular switch between fibroblast necrosis and apoptosis in death receptor signaling.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

The mitochondrial permeability transition.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

NAD+ repletion prevents PARP-1-induced glycolytic blockade and cell death in cultured mouse astrocytes

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M508135200

21 January 2018

Identifiers

DOI

10.1074/JBC.M508135200

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19 September 2017

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