NF-kappaB activation but not PI3K/Akt is required for dexamethasone dependent protection against TNF-alpha cytotoxicity in L929 cells (Q40401378)

20 September 2017

title

NF-kappaB activation but not PI3K/Akt is required for dexamethasone dependent protection against TNF-alpha cytotoxicity in L929 cells (English)

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20 September 2017

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Criselda Mendoza-Milla

1

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20 September 2017

Catalina Machuca Rodríguez

2

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20 September 2017

Emilio Córdova Alarcón

3

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20 September 2017

Adriana Estrada Bernal

4

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20 September 2017

E Mayra Toledo-Cuevas

5

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20 September 2017

Eduardo Martínez Martínez

6

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20 September 2017

Alejandro Zentella Dehesa

7

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20 September 2017

language of work or name

English

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publication date

1 July 2005

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20 September 2017

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20 September 2017

volume

579

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20 September 2017

issue

18

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20 September 2017

page(s)

3947-3952

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The TNF receptor 1-associated protein TRADD signals cell death and NF-kappa B activation

20 September 2017

cites work

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Tumor necrosis factor-alpha-induced cell killing and activation of transcription factor NF-kappaB are uncoupled in L929 cells

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NF-kappaB at the crossroads of life and death

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Phosphorylation meets ubiquitination: the control of NF-[kappa]B activity

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NF-kappaB antiapoptosis: induction of TRAF1 and TRAF2 and c-IAP1 and c-IAP2 to suppress caspase-8 activation

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Akt phosphorylation of BAD couples survival signals to the cell-intrinsic death machinery

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PI3-K/AKT regulation of NF-kappaB signaling events in suppression of TNF-induced apoptosis

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NF-kappaB activation by tumour necrosis factor requires the Akt serine-threonine kinase

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An essential role for NF-kappaB in preventing TNF-alpha-induced cell death

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Ceramide reproduces late appearance of oxidative stress during TNF-mediated cell death in L929 cells

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Ceramide promotes the death of human cervical tumor cells in the absence of biochemical and morphological markers of apoptosis

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1-Phosphatidylinositol 3-kinase activity is required for insulin-stimulated glucose transport but not for RAS activation in CHO cells

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16K prolactin induces NF-kappaB activation in pulmonary fibroblasts

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A phosphatidylinositol 3-kinase/Akt pathway, activated by tumor necrosis factor or interleukin-1, inhibits apoptosis but does not activate NFkappaB in human endothelial cells

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Constitutively activated Akt-1 is vital for the survival of human monocyte-differentiated macrophages. Role of Mcl-1, independent of nuclear factor (NF)-kappaB, Bad, or caspase activation.

21 January 2018

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Cell type-specific expression of the IkappaB kinases determines the significance of phosphatidylinositol 3-kinase/Akt signaling to NF-kappa B activation

21 January 2018

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Reduction in tumor necrosis factor receptor affinity and cytotoxicity by glucocorticoids

21 January 2018

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Identification of cytokine-induced nuclear factor-kappaB target genes in ovarian and breast cancer cells

21 January 2018

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10.1016/J.FEBSLET.2005.05.081

21 January 2018

Identifiers

DOI

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20 September 2017

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