The transcriptome of HCV replicon expressing cell lines in the presence of alpha interferon (Q40433212)

20 September 2017

The transcriptome of HCV replicon expressing cell lines in the presence of alpha interferon (English)

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20 September 2017

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Junpei Hayashi

1

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20 September 2017

Radka Stoyanova

series ordinal

2

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20 September 2017

Christoph Seeger

series ordinal

3

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20 September 2017

language of work or name

English

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publication date

1 May 2005

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20 September 2017

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20 September 2017

335

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20 September 2017

2

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20 September 2017

264-275

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Physical and functional interaction between HCV core protein and the different p73 isoforms

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Hepatitis C virus core protein promotes cell proliferation through the upregulation of cyclin E expression levels.

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Human hepatitis C virus NS5A protein alters intracellular calcium levels, induces oxidative stress, and activates STAT-3 and NF-kappa B.

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Effect of alpha interferon on the hepatitis C virus replicon

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Cytopathic and Noncytopathic Interferon Responses in Cells Expressing Hepatitis C Virus Subgenomic Replicons

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Modulation of p53 transcription regulatory activity and post-translational modification by hepatitis C virus core protein

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Impaired antiviral response in human hepatoma cells.

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p53-dependent transcriptional repression of p21(waf1) by hepatitis C virus NS3.

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HCV NS5A interacts with p53 and inhibits p53-mediated apoptosis

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Involvement of proteasomes in gene induction by interferon and double-stranded RNA.

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Activation of p53 tumor suppressor by hepatitis C virus core protein

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Hepatitis C virus NS5A: tales of a promiscuous protein

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Hepatitis C virus core protein expression leads to biphasic regulation of the p21 cdk inhibitor and modulation of hepatocyte cell cycle

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Hepatitis C virus core functions as a suppressor of cyclin-dependent kinase-activating kinase and impairs cell cycle progression

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Hepatitis C virus core protein enhances p53 function through augmentation of DNA binding affinity and transcriptional ability

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Cell-type and donor-specific transcriptional responses to interferon-alpha. Use of customized gene arrays.

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NS5A, a nonstructural protein of hepatitis C virus, binds growth factor receptor-bound protein 2 adaptor protein in a Src homology 3 domain/ligand-dependent manner and perturbs mitogenic signaling

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The RNA helicase RIG-I has an essential function in double-stranded RNA-induced innate antiviral responses

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10.1016/J.VIROL.2005.02.018

7 January 2021

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DOI

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20 September 2017

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