Human Parvovirus B19 nonstructural protein transactivates the p21/WAF1 through Sp1. (Q40497654)

21 September 2017

Human Parvovirus B19 nonstructural protein transactivates the p21/WAF1 through Sp1. (English)

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21 September 2017

Akitoshi Nakashima

1

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21 September 2017

Eiji Morita

series ordinal

2

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21 September 2017

Shigeru Saito

series ordinal

3

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21 September 2017

Kazuo Sugamura

series ordinal

4

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21 September 2017

language of work or name

English

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publication date

1 November 2004

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21 September 2017

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21 September 2017

329

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21 September 2017

2

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21 September 2017

493-504

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Down-regulation of human type II collagen gene expression by transforming growth factor-beta 1 (TGF-beta 1) in articular chondrocytes involves SP3/SP1 ratio

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Sp1-mediated transcriptional activation is repressed by Sp3

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Transcriptional activation by the parvoviral nonstructural protein NS-1 is mediated via a direct interaction with Sp1

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Human parvovirus B19 induces cell cycle arrest at G(2) phase with accumulation of mitotic cyclins

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Human parvovirus B19 nonstructural protein (NS1) induces cell cycle arrest at G(1) phase

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Butyrate activates the WAF1/Cip1 gene promoter through Sp1 sites in a p53-negative human colon cancer cell line

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NS1 protein of parvovirus B19 interacts directly with DNA sequences of the p6 promoter and with the cellular transcription factors Sp1/Sp3.

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p300 collaborates with Sp1 and Sp3 in p21(waf1/cip1) promoter activation induced by histone deacetylase inhibitor

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Parvovirus B19 infection induces apoptosis of erythroid cells in vitro and in vivo

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The GC box and TATA transcription control elements in the P38 promoter of the minute virus of mice are necessary and sufficient for transactivation by the nonstructural protein NS1.

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inferred from DOI database lookup

Characterization of cis-acting and NS1 protein-responsive elements in the p6 promoter of parvovirus B19.

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2004.09.008

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The NS1 protein of the autonomous parvovirus minute virus of mice blocks cellular DNA replication: a consequence of lesions to the chromatin?

1 reference

https://api.crossref.org/works/10.1016%2FJ.VIROL.2004.09.008

7 January 2021

inferred from DOI database lookup

Identifiers

DOI

10.1016/J.VIROL.2004.09.008

1 reference

release_sw4wfrwlpbbx7mphmido5fpo6y

21 September 2017

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Fatcat

https://api.fatcat.wiki/v0/release/sw4wfrwlpbbx7mphmido5fpo6y

24 November 2022

mapped directly with Wikidata item

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