Direct evidence for the absence of intercellular trafficking of VP22 fused to GFP or to the herpes simplex virus thymidine kinase. (Q40504496)

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4 January 2020

title

Direct evidence for the absence of intercellular trafficking of VP22 fused to GFP or to the herpes simplex virus thymidine kinase (English)

1 reference

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4 January 2020

author

Pedro O de Campos-Lima

3

1 reference

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4 January 2020

Manuel Caruso

4

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4 January 2020

author name string

Roy V

1

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4 January 2020

Qiao J

2

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4 January 2020

publication date

1 January 2005

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4 January 2020

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4 January 2020

volume

12

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4 January 2020

issue

2

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4 January 2020

page(s)

169-176

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Therapy of malignant brain tumors by intratumoral implantation of retroviral vector-producing cells

4 January 2020

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Bystander killing of cancer cells by herpes simplex virus thymidine kinase gene is mediated by connexins

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Retinoids augment the bystander effect in vitro and in vivo in herpes simplex virus thymidine kinase/ganciclovir-mediated gene therapy

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The bystander effect exerted by tumor cells expressing the herpes simplex virus thymidine kinase (HSVtk) gene is dependent on connexin expression and cell communication via gap junctions

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Enhancement of the herpes simplex virus thymidine kinase/ganciclovir bystander effect and its antitumor efficacy in vivo by pharmacologic manipulation of gap junctions

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Pharmacological modulation of the bystander effect in the herpes simplex virus thymidine kinase/ganciclovir gene therapy system: effects of dibutyryl adenosine 3',5'-cyclic monophosphate, alpha-glycyrrhetinic acid, and cytosine arabinoside

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Retrovirus-mediated transfer of the herpes simplex virus thymidine kinase and connexin26 genes in pancreatic cells results in variable efficiency on the bystander killing: implications for gene therapy

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Intercellular delivery of thymidine kinase prodrug activating enzyme by the herpes simplex virus protein, VP22.

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VP22 enhanced intercellular trafficking of HSV thymidine kinase reduced the level of ganciclovir needed to cause suicide cell death

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Intercellular trafficking and protein delivery by a herpesvirus structural protein

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Intercellular delivery of functional p53 by the herpesvirus protein VP22.

21 January 2018

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Intratumoral spread and increased efficacy of a p53-VP22 fusion protein expressed by a recombinant adenovirus

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Is VP22 nuclear homing an artifact?

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Full-length proteins attached to the HIV tat protein transduction domain are neither transduced between cells, nor exhibit enhanced immunogenicity

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21 January 2018

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Protein delivery using VP22.

21 January 2018

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Different thermostabilities of FLP and Cre recombinases: implications for applied site-specific recombination

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21 January 2018

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High-titer packaging cells producing recombinant retroviruses resistant to human serum

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inferred from DOI database lookup

7 January 2021

based on heuristic

Ability of the Tat basic domain and VP22 to mediate cell binding, but not membrane translocation of the diphtheria toxin A-fragment

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inferred from DOI database lookup

7 January 2021

based on heuristic

Positively charged DNA-binding proteins cause apparent cell membrane translocation

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inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1038/SJ.GT.3302394

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:15483667%20AND%20SRC:MED&resulttype=core&format=json

Dimensions Publication ID

4 January 2020

1014355978

0 references

1 reference