BZLF1, an Epstein-Barr virus immediate-early protein, induces p65 nuclear translocation while inhibiting p65 transcriptional function (Q40508322)

21 September 2017

BZLF1, an Epstein-Barr virus immediate-early protein, induces p65 nuclear translocation while inhibiting p65 transcriptional function (English)

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21 September 2017

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Thomas E Morrison

1

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PubMed ID

15464842

21 September 2017

Shannon C Kenney

series ordinal

2

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PubMed ID

15464842

21 September 2017

language of work or name

English

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publication date

1 October 2004

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21 September 2017

14

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inferred from page(s)

published in

Virology

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21 September 2017

328

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21 September 2017

2

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21 September 2017

219-232

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Control of Epstein-Barr virus reactivation by activated CD40 and viral latent membrane protein 1.

21 September 2017

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Identification by in vivo genomic footprinting of a transcriptional switch containing NF-kappaB and Sp1 that regulates the IkappaBalpha promoter

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Inducible nuclear expression of newly synthesized I kappa B alpha negatively regulates DNA-binding and transcriptional activities of NF-kappa B.

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Series introduction: the transcription factor NF-kappaB and human disease

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Autoregulation of I kappa B alpha activity

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The CBP bromodomain and nucleosome targeting are required for Zta-directed nucleosome acetylation and transcription activation

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Accurate transcription initiation by RNA polymerase II in a soluble extract from isolated mammalian nuclei

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DNA-binding-defective mutants of the Epstein-Barr virus lytic switch activator Zta transactivate with altered specificities

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The bZIP transactivator of Epstein-Barr virus, BZLF1, functionally and physically interacts with the p65 subunit of NF-kappa B

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Phosphorylation meets ubiquitination: the control of NF-[kappa]B activity

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The lytic cycle of Epstein-Barr virus is associated with decreased expression of cell surface major histocompatibility complex class I and class II molecules

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Cytokine-inducible CD40 gene expression in vascular smooth muscle cells is mediated by nuclear factor kappaB and signal transducer and activation of transcription-1

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IKK-1 and IKK-2: cytokine-activated IkappaB kinases essential for NF-kappaB activation

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Epstein-Barr virus immediate-early protein BZLF1 inhibits tumor necrosis factor alpha-induced signaling and apoptosis by downregulating tumor necrosis factor receptor 1

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The glucocorticoid receptor inhibits NFkappaB by interfering with serine-2 phosphorylation of the RNA polymerase II carboxy-terminal domain

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Herpes simplex type 1 induction of persistent NF-kappa B nuclear translocation increases the efficiency of virus replication

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Latent membrane protein 1 inhibits Epstein-Barr virus lytic cycle induction and progress via different mechanisms.

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p38-Dependent marking of inflammatory genes for increased NF-kappa B recruitment

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Activation of phosphatidylinositol 3-kinase in response to interleukin-1 leads to phosphorylation and activation of the NF-kappaB p65/RelA subunit

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Distinct roles of the Ikappa B kinase alpha and beta subunits in liberating nuclear factor kappa B (NF-kappa B) from Ikappa B and in phosphorylating the p65 subunit of NF-kappa B.

7 January 2021

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Functional and physical associations between NF-kappa B and C/EBP family members: a Rel domain-bZIP interaction

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Cytokine-inducible CD40 expression in human endothelial cells is mediated by interferon regulatory factor-1.

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IKK beta plays an essential role in the phosphorylation of RelA/p65 on serine 536 induced by lipopolysaccharide

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Epstein-Barr viral latency is disrupted by the immediate-early BRLF1 protein through a cell-specific mechanism

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The IkappaB kinase complex (IKK) contains two kinase subunits, IKKalpha and IKKbeta, necessary for IkappaB phosphorylation and NF-kappaB activation

7 January 2021

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The amino-terminal C/H1 domain of CREB binding protein mediates zta transcriptional activation of latent Epstein-Barr virus

7 January 2021

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The Epstein-Barr virus BZLF1 protein interacts physically and functionally with the histone acetylase CREB-binding protein

7 January 2021

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7 January 2021

inferred from DOI database lookup

Characterization of the Epstein-Barr virus BZLF1 protein transactivation domain

1 reference

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7 January 2021

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Responsiveness of the Epstein-Barr virus NotI repeat promoter to the Z transactivator is mediated in a cell-type-specific manner by two independent signal regions

1 reference

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7 January 2021

inferred from DOI database lookup

Identifiers

DOI

10.1016/J.VIROL.2004.07.020

1 reference

21 September 2017

1 reference

PubMed ID

15464842