Statins inhibit reoxygenation-induced cardiomyocyte apoptosis: role for glycogen synthase kinase 3beta and transcription factor beta-catenin (Q40518725)

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scientific article published on September 2004

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English	Statins inhibit reoxygenation-induced cardiomyocyte apoptosis: role for glycogen synthase kinase 3beta and transcription factor beta-catenin	scientific article published on September 2004

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scholarly article

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Europe PubMed Central

21 September 2017

Statins inhibit reoxygenation-induced cardiomyocyte apoptosis: role for glycogen synthase kinase 3beta and transcription factor beta-catenin (English)

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Europe PubMed Central

21 September 2017

Anthony Baurand

4

object named as

Anthony Baurand

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Europe PubMed Central

21 September 2017

author name string

Martin W Bergmann

1

1 reference

Europe PubMed Central

21 September 2017

Cindy Rechner

2

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Europe PubMed Central

21 September 2017

Christian Freund

3

1 reference

Europe PubMed Central

21 September 2017

Amina El Jamali

5

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Europe PubMed Central

21 September 2017

Rainer Dietz

6

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Europe PubMed Central

21 September 2017

publication date

1 September 2004

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Europe PubMed Central

21 September 2017

Journal of Molecular and Cellular Cardiology

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Europe PubMed Central

21 September 2017

37

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Europe PubMed Central

21 September 2017

3

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Europe PubMed Central

21 September 2017

681-690

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Europe PubMed Central

21 September 2017

Improvement of left ventricular remodeling and function by hydroxymethylglutaryl coenzyme a reductase inhibition with cerivastatin in rats with heart failure after myocardial infarction

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Fluvastatin, a 3-hydroxy-3-methylglutaryl coenzyme a reductase inhibitor, attenuates left ventricular remodeling and failure after experimental myocardial infarction

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Simvastatin preserves the ischemic-reperfused myocardium in normocholesterolemic rat hearts

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The HMG-CoA reductase inhibitor simvastatin activates the protein kinase Akt and promotes angiogenesis in normocholesterolemic animals

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HMG-CoA reductase inhibitors (statins) increase endothelial progenitor cells via the PI 3-kinase/Akt pathway

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Regulation of cardiomyocyte apoptotic signaling by insulin-like growth factor I.

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In cardiomyocyte hypoxia, insulin-like growth factor-I-induced antiapoptotic signaling requires phosphatidylinositol-3-OH-kinase-dependent and mitogen-activated protein kinase-dependent activation of the transcription factor cAMP response element-bi

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Myocardial protection by insulin at reperfusion requires early administration and is mediated via Akt and p70s6 kinase cell-survival signaling

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Adenoviral gene transfer of activated phosphatidylinositol 3'-kinase and Akt inhibits apoptosis of hypoxic cardiomyocytes in vitro

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Intracoronary, adenovirus-mediated Akt gene transfer in heart limits infarct size following ischemia-reperfusion injury in vivo

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Phosphorylation of glycogen synthase kinase-3beta during preconditioning through a phosphatidylinositol-3-kinase--dependent pathway is cardioprotective

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The integrin-linked kinase regulates the cyclin D1 gene through glycogen synthase kinase 3beta and cAMP-responsive element-binding protein-dependent pathways.

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Glycogen synthase kinase 3beta-mediated apoptosis of primary cortical astrocytes involves inhibition of nuclear factor kappaB signaling

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Akt suppresses apoptosis by stimulating the transactivation potential of the RelA/p65 subunit of NF-kappaB.

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Stabilization of beta-catenin by a Wnt-independent mechanism regulates cardiomyocyte growth

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Effect of NF-kappa B Inhibition on TNF-alpha-induced apoptosis and downstream pathways in cardiomyocytes

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A mammalian myocardial cell-free system to study cell cycle reentry in terminally differentiated cardiomyocytes

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Rhodamine 110-Linked Amino Acids and Peptides as Substrates To Measure Caspase Activity upon Apoptosis Induction in Intact Cells†

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Fas receptor signaling inhibits glycogen synthase kinase 3 beta and induces cardiac hypertrophy following pressure overload

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Direct, activating interaction between glycogen synthase kinase-3beta and p53 after DNA damage

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Morphological and molecular characterization of adult cardiomyocyte apoptosis during hypoxia and reoxygenation

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HMG-CoA reductase inhibitor mobilizes bone marrow--derived endothelial progenitor cells

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Direct vascular and cardioprotective effects of rosuvastatin, a new HMG-CoA reductase inhibitor

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Atorvastatin, administered at the onset of reperfusion, and independent of lipid lowering, protects the myocardium by up-regulating a pro-survival pathway

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Reduction of myocardial infarct size by fluvastatin

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Suppression of endothelial nitric oxide production after withdrawal of statin treatment is mediated by negative feedback regulation of rho GTPase gene transcription.

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Double-edged role of statins in angiogenesis signaling

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Expression of secreted frizzled related proteins 3 and 4 in human ventricular myocardium correlates with apoptosis related gene expression

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Enhanced glycogen synthase kinase-3beta activity mediates hypoxia-induced apoptosis of vascular smooth muscle cells and is prevented by glucose transport and metabolism

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Essential myosin light chain as a target for caspase-3 in failing myocardium

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Simvastatin attenuates oxidant-induced mitochondrial dysfunction in cardiac myocytes

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Are lipid-lowering drugs also antiarrhythmic drugs? An analysis of the Antiarrhythmics versus Implantable Defibrillators (AVID) trial

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Impact of HMG CoA reductase inhibition on small GTPases in the heart

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Statins as antioxidant therapy for preventing cardiac myocyte hypertrophy

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Nerve growth factor signals through TrkA, phosphatidylinositol 3-kinase, and Rac1 to inactivate RhoA during the initiation of neuronal differentiation of PC12 cells

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CREB is a regulatory target for the protein kinase Akt/PKB

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A Wnt- and beta -catenin-dependent pathway for mammalian cardiac myogenesis

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A role for the beta-catenin/T-cell factor signaling cascade in vascular remodeling

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Fibroblast growth factor 1 regulates signaling via the glycogen synthase kinase-3beta pathway. Implications for neuroprotection

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Preprocedural statin medication reduces the extent of periprocedural non-Q-wave myocardial infarction

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Activated glycogen synthase-3 beta suppresses cardiac hypertrophy in vivo

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Identifiers

10.1016/J.YJMCC.2004.05.025

1 reference

Europe PubMed Central

21 September 2017

1 reference

Europe PubMed Central

21 September 2017

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