Advanced glycosylation end products induce NF-kappaB dependent iNOS expression in RAW 264.7 cells (Q40702746)

22 September 2017

Advanced glycosylation end products induce NF-kappaB dependent iNOS expression in RAW 264.7 cells (English)

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22 September 2017

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Chih-Hsiung Wu

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22 September 2017

Chao-Ming Huang

2

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22 September 2017

Chien-Huang Lin

3

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22 September 2017

Yuan-Sun Ho

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22 September 2017

Chien-Ming Chen

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22 September 2017

Horng-Mo Lee

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22 September 2017

1 August 2002

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Molecular and Cellular Endocrinology

22 September 2017

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22 September 2017

194

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22 September 2017

9-17

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22 September 2017

1-2

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Nonenzymatically glycated albumin (Amadori adducts) enhances nitric oxide synthase activity and gene expression in endothelial cells

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Oxidative stress and nuclear factor-kappaB activation: a reassessment of the evidence in the light of recent discoveries.

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Control of I kappa B-alpha proteolysis by site-specific, signal-induced phosphorylation

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Glycosylation products as toxic mediators of diabetic complications

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Signal-induced site-specific phosphorylation targets I kappa B alpha to the ubiquitin-proteasome pathway

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Epstein-Barr virus envelope glycoprotein gp350 induces NF-kappaB activation and IL-1beta synthesis in human monocytes-macrophages involving PKC and PI3-K.

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Investigations on oxidative stress and therapeutical implications in dementia

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Cytokine-activated endothelial cells express an isotype of nitric oxide synthase which is tetrahydrobiopterin-dependent, calmodulin-independent and inhibited by arginine analogs with a rank-order of potency characteristic of activated macrophages

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LPS induction of gene expression in human monocytes

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Possible role of tumor necrosis factor and interleukin-1 in the development of diabetic nephropathy

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The receptor for advanced glycation end-products has a central role in mediating the effects of advanced glycation end-products on the development of vascular disease in diabetes mellitus

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Receptor for advanced glycation end products (RAGE)-mediated neurite outgrowth and activation of NF-kappaB require the cytoplasmic domain of the receptor but different downstream signaling pathways

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Analysis of the signal transduction in the induction of nitric oxide synthase by lipoteichoic acid in macrophages

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Effect of Advanced Glycation End Product–Modified Albumin on Tissue Factor Expression by Monocytes

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Activation of the receptor for advanced glycation end products triggers a p21(ras)-dependent mitogen-activated protein kinase pathway regulated by oxidant stress.

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Molecular identity and cellular distribution of advanced glycation endproduct receptors: relationship of p60 to OST-48 and p90 to 80K-H membrane proteins.

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Activation of nuclear factor kappaB and induction of inducible nitric oxide synthase by Ureaplasma urealyticum in macrophages

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Advanced glycosylation end products induce nitric oxide synthase expression in C6 glioma cells: involvement of a p38 MAP kinase-dependent mechanism

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Cytotoxicity of advanced glycation endproducts is mediated by oxidative stress

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The role of oxidative stress and NF-kappaB activation in late diabetic complications

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Alzheimer's disease--synergistic effects of glucose deficit, oxidative stress and advanced glycation endproducts

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High molecular weight hyaluronic acid inhibits advanced glycation endproduct-induced NF-kappaB activation and cytokine expression.

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Ultrastructural changes and immunohistochemical localization of nitric oxide synthase, advanced glycation end products and NF-kappa B in aorta of streptozotocin treated Mongolian gerbils

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Endothelin 1 transcription is controlled by nuclear factor-kappaB in AGE-stimulated cultured endothelial cells

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Effect of advanced glycosylation end products on the induction of nitric oxide synthase in murine macrophages

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Regulation of human mononuclear phagocyte migration by cell surface-binding proteins for advanced glycation end products

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Advanced glycation endproducts (AGEs) induce oxidant stress in the gingiva: a potential mechanism underlying accelerated periodontal disease associated with diabetes.

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Advanced glycation endproducts stimulate the MAP-kinase pathway in tubulus cell line LLC-PK1.

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Human and rat mesangial cell receptors for glucose-modified proteins: potential role in kidney tissue remodelling and diabetic nephropathy

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The receptor for advanced glycation end products is induced by the glycation products themselves and tumor necrosis factor-alpha through nuclear factor-kappa B, and by 17beta-estradiol through Sp-1 in human vascular endothelial cells

1 reference

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Receptor-mediated endothelial cell dysfunction in diabetic vasculopathy. Soluble receptor for advanced glycation end products blocks hyperpermeability in diabetic rats

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HTLV-I Tax protein binds to MEKK1 to stimulate IkappaB kinase activity and NF-kappaB activation.

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7 January 2021

10.1016/S0303-7207(02)00212-5

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22 September 2017

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