Advanced glycosylation end products induce NF-kappaB dependent iNOS expression in RAW 264.7 cells (Q40702746)

22 September 2017

title

Advanced glycosylation end products induce NF-kappaB dependent iNOS expression in RAW 264.7 cells (English)

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22 September 2017

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Chih-Hsiung Wu

1

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22 September 2017

Chao-Ming Huang

2

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22 September 2017

Chien-Huang Lin

3

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22 September 2017

Yuan-Sun Ho

4

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22 September 2017

Chien-Ming Chen

5

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22 September 2017

Horng-Mo Lee

6

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22 September 2017

publication date

1 August 2002

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Molecular and Cellular Endocrinology

22 September 2017

published in

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22 September 2017

volume

194

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22 September 2017

issue

1-2

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22 September 2017

page(s)

9-17

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Nonenzymatically glycated albumin (Amadori adducts) enhances nitric oxide synthase activity and gene expression in endothelial cells

22 September 2017

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The receptor for advanced glycation end-products has a central role in mediating the effects of advanced glycation end-products on the development of vascular disease in diabetes mellitus

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Effect of Advanced Glycation End Product–Modified Albumin on Tissue Factor Expression by Monocytes

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Activation of the receptor for advanced glycation end products triggers a p21(ras)-dependent mitogen-activated protein kinase pathway regulated by oxidant stress.

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Advanced glycosylation end products induce nitric oxide synthase expression in C6 glioma cells: involvement of a p38 MAP kinase-dependent mechanism

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The role of oxidative stress and NF-kappaB activation in late diabetic complications

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Alzheimer's disease--synergistic effects of glucose deficit, oxidative stress and advanced glycation endproducts

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7 January 2021

High molecular weight hyaluronic acid inhibits advanced glycation endproduct-induced NF-kappaB activation and cytokine expression

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7 January 2021

The receptor for advanced glycation end products is induced by the glycation products themselves and tumor necrosis factor-alpha through nuclear factor-kappa B, and by 17beta-estradiol through Sp-1 in human vascular endothelial cells

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7 January 2021

Receptor-mediated endothelial cell dysfunction in diabetic vasculopathy. Soluble receptor for advanced glycation end products blocks hyperpermeability in diabetic rats

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HTLV-I Tax protein binds to MEKK1 to stimulate IkappaB kinase activity and NF-kappaB activation

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Identifiers

DOI

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22 September 2017

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