Calretinin and calbindin D-28k, but not parvalbumin protect against glutamate-induced delayed excitotoxicity in transfected N18-RE 105 neuroblastoma-retina hybrid cells. (Q40717215)

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scientific article published on August 2002

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English	Calretinin and calbindin D-28k, but not parvalbumin protect against glutamate-induced delayed excitotoxicity in transfected N18-RE 105 neuroblastoma-retina hybrid cells.	scientific article published on August 2002

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11 November 2019

Calretinin and calbindin D-28k, but not parvalbumin protect against glutamate-induced delayed excitotoxicity in transfected N18-RE 105 neuroblastoma-retina hybrid cells (English)

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11 November 2019

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11 November 2019

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C D'Orlando

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11 November 2019

M R Celio

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1 August 2002

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11 November 2019

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11 November 2019

Ataxia and altered dendritic calcium signaling in mice carrying a targeted null mutation of the calbindin D28k gene

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L-glutamate binding site on N18-RE-105 neuroblastoma hybrid cells is not coupled to an ion channel

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Calretinin and calbindin D-28k delay the onset of cell death after excitotoxic stimulation in transfected P19 cells

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Relative resistance of striatal neurons containing calbindin or parvalbumin to quinolinic acid-mediated excitotoxicity compared to other striatal neuron types.

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The calcium-binding protein calretinin-22k, an alternative splicing product of the calretinin gene is expressed in several colon adeno carcinoma cell lines

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Expression of the calcium-binding protein, parvalbumin, in cultured cortical neurons using a HSV-1 vector system enhances NMDA neurotoxicity.

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Calbindin28kDa and calbindin30kDa (calretinin) are substantially localised in the particulate fraction of rat brain

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Calbindin-D28K-containing neurons in animal models of neurodegeneration: possible protection from excitotoxicity

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Calretinin-immunoreactive dopaminergic neurons from embryonic rat mesencephalon are resistant to levodopa-induced neurotoxicity

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Vulnerability to calcium-induced neurotoxicity in cultured neurons expressing calretinin.

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Calbindin-D28k fails to protect hippocampal neurons against ischemia in spite of its cytoplasmic calcium buffering properties: evidence from calbindin-D28k knockout mice.

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The expression of calretinin in transfected PC12 cells provides no protection against Ca(2+)-overload or trophic factor deprivation

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Kinetics of Ca2+ binding to parvalbumin in bovine chromaffin cells: implications for [Ca2+] transients of neuronal dendrites

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Cortical neurons containing calretinin are selectively resistant to calcium overload and excitotoxicity in vitro.

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Glutamate toxicity in a neuronal cell line involves inhibition of cystine transport leading to oxidative stress

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Glutamate cytotoxicity in a neuronal cell line is blocked by membrane depolarization

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Usefulness and limitations of linear approximations to the understanding of Ca++ signals

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GABAergic hippocampal neurons resistant to ischemia-induced neuronal death contain the Ca2(+)-binding protein parvalbumin

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Three types of neuronal calcium channel with different calcium agonist sensitivity.

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Calbindin-D28K and ischemic damage of pyramidal cells in rat hippocampus.

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Calcium Dependence of Toxic Cell Death: A Final Common Pathway

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1 reference

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1 reference

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1 reference

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1 reference

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Characteristics of chloride-dependent incorporation of glutamate into brain membranes argue against a receptor binding site

1 reference

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Quisqualate-sensitive, chloride-dependent transport of glutamate into rat brain synaptosomes.

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Identifiers

10.1016/S0006-8993(02)02753-1

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Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:12126880%20AND%20SRC:MED&resulttype=core&format=json

11 November 2019

1 reference

Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:12126880%20AND%20SRC:MED&resulttype=core&format=json

11 November 2019

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