Mechanism of membrane depolarization caused by the Alzheimer Abeta1-42 peptide (Q40726766)

23 September 2017

Mechanism of membrane depolarization caused by the Alzheimer Abeta1-42 peptide (English)

1 reference

23 September 2017

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Barbara J Blanchard

1

1 reference

PubMed ID

12054502

23 September 2017

Veena L Thomas

series ordinal

2

1 reference

PubMed ID

12054502

23 September 2017

Vernon M Ingram

series ordinal

3

1 reference

23 September 2017

1 May 2002

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Biochemical and Biophysical Research Communications

23 September 2017

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23 September 2017

293

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23 September 2017

1197-1203

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23 September 2017

4

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Clearing the brain's amyloid cobwebs

23 September 2017

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900346-7

Structural model for the beta-amyloid fibril based on interstrand alignment of an antiparallel-sheet comprising a C-terminal peptide

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900346-7

Mechanism and prevention of neurotoxicity caused by beta-amyloid peptides: relation to Alzheimer's disease

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900346-7

The toxicity in vitro of beta-amyloid protein

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900346-7

beta-Amyloid peptides destabilize calcium homeostasis and render human cortical neurons vulnerable to excitotoxicity

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900346-7

beta-Amyloid precursor protein metabolites and loss of neuronal Ca2+ homeostasis in Alzheimer's disease.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900346-7

Calcium-destabilizing and neurodegenerative effects of aggregated beta-amyloid peptide are attenuated by basic FGF.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900346-7

Compromised mitochondrial function leads to increased cytosolic calcium and to activation of MAP kinases

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900346-7

Use of oxonol V as a probe of membrane potential in proteoliposomes containing cytochrome oxidase in the submitochondrial orientation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900346-7

Hyperpolarization of isolated capillaries from guinea-pig heart induced by K+ channel openers and glucose deprivation

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900346-7

An improved method of preparing the amyloid beta-protein for fibrillogenesis and neurotoxicity experiments.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900346-7

Protein tyrosine kinase is required for the induction of long-term potentiation in the rat hippocampus

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900346-7

Long-term potentiation in the hippocampus is blocked by tyrosine kinase inhibitors

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900346-7

Epidermal-growth-factor-dependent activation of the src-family kinases

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900346-7

Eliminating membrane depolarization caused by the Alzheimer peptide Abeta(1-42, aggr.).

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900346-7

Attenuation of beta-amyloid neurotoxicity in vitro by potassium-induced depolarization

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2802%2900346-7

inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1016/S0006-291X(02)00346-7

1 reference

23 September 2017

1 reference

PubMed ID

12054502