Protein kinase C and ERK activation are required for TFF-peptide-stimulated bronchial epithelial cell migration and tumor necrosis factor-alpha-induced interleukin-6 (IL-6) and IL-8 secretion (Q40746058)

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scientific article published on 7 March 2002

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English	Protein kinase C and ERK activation are required for TFF-peptide-stimulated bronchial epithelial cell migration and tumor necrosis factor-alpha-induced interleukin-6 (IL-6) and IL-8 secretion	scientific article published on 7 March 2002

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scholarly article

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Europe PubMed Central

PubMed publication ID

23 September 2017

Protein kinase C and ERK activation are required for TFF-peptide-stimulated bronchial epithelial cell migration and tumor necrosis factor-alpha-induced interleukin-6 (IL-6) and IL-8 secretion (English)

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

author name string

Angela Graness

1

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23 September 2017

Caroline E Chwieralski

2

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23 September 2017

Dirk Reinhold

3

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23 September 2017

Lars Thim

4

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23 September 2017

Werner Hoffmann

5

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23 September 2017

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publication date

7 March 2002

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23 September 2017

Journal of Biological Chemistry

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23 September 2017

277

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23 September 2017

21

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Europe PubMed Central

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23 September 2017

18440-18446

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PubMed publication ID

23 September 2017

Cell type specific expression of secretory TFF peptides: colocalization with mucins and synthesis in the brain

1 reference

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Trefoil peptides: from structure to function

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Rolling in the clover: trefoil factor family (TFF)-domain peptides, cell migration and cancer.

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Spasmolytic polypeptide is a major antral peptide: distribution of the trefoil peptides human spasmolytic polypeptide and pS2 in the stomach

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Aspects of the biology of regeneration and repair in the human gastrointestinal tract.

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pS2/TFF1 interacts directly with the VWFC cysteine-rich domains of mucins

1 reference

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Trefoil factor family-peptides promote migration of human bronchial epithelial cells: synergistic effect with epidermal growth factor

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Transcription factor NF-kappaB signals antianoikic function of trefoil factor 3 on intestinal epithelial cells

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Intestinal trefoil factor confers colonic epithelial resistance to apoptosis

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Distinct pathways of cell migration and antiapoptotic response to epithelial injury: structure-function analysis of human intestinal trefoil factor

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Regulation of interleukin-8 gene expression

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hP1.B, a human P-domain peptide homologous with rat intestinal trefoil factor, is expressed also in the ulcer-associated cell lineage and the uterus

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Characterization of human and rat intestinal trefoil factor produced in yeast

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A Novel Mitogenic Signaling Pathway of Bradykinin in the Human Colon Carcinoma Cell Line SW-480 Involves Sequential Activation of a Gq/11 Protein, Phosphatidylinositol 3-Kinase , and Protein Kinase C

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Novel bradykinin signalling events in PC-12 cells: stimulation of the cAMP pathway leads to cAMP-mediated translocation of protein kinase Cepsilon

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The pathophysiology of tumor necrosis factors

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Two tumour necrosis factor receptors: structure and function.

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Dissection of TNF receptor 1 effector functions: JNK activation is not linked to apoptosis while NF-kappaB activation prevents cell death

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Protein kinase cascades activated by stress and inflammatory cytokines

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Tumor necrosis factor signaling to stress-activated protein kinase (SAPK)/Jun NH2-terminal kinase (JNK) and p38. Germinal center kinase couples TRAF2 to mitogen-activated protein kinase/ERK kinase kinase 1 and SAPK while receptor interacting [...]

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Engagement of tumor necrosis factor (TNF) receptor 1 leads to ATF-2- and p38 mitogen-activated protein kinase-dependent TNF-alpha gene expression

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Cytokine signal transduction

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Glucocorticoids repress NF-kappaB-driven genes by disturbing the interaction of p65 with the basal transcription machinery, irrespective of coactivator levels in the cell

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Induction of interleukin-8 synthesis integrates effects on transcription and mRNA degradation from at least three different cytokine- or stress-activated signal transduction pathways

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A promoter recruitment mechanism for tumor necrosis factor-alpha-induced interleukin-8 transcription in type II pulmonary epithelial cells. Dependence on nuclear abundance of Rel A, NF-kappaB1, and c-Rel transcription factors

1 reference

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H2O2 and tumor necrosis factor-alpha induce differential binding of the redox-responsive transcription factors AP-1 and NF-kappaB to the interleukin-8 promoter in endothelial and epithelial cells

1 reference

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Signal transduction pathways mediating neurotensin-stimulated interleukin-8 expression in human colonocytes

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The regulation of AP-1 activity by mitogen-activated protein kinases

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Phosphorylation at threonine-235 by a ras-dependent mitogen-activated protein kinase cascade is essential for transcription factor NF-IL6

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Transcription factors NF-IL6 and NF-kappa B synergistically activate transcription of the inflammatory cytokines, interleukin 6 and interleukin 8

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Myosin ii light chain phosphorylation regulates membrane localization and apoptotic signaling of tumor necrosis factor receptor-1

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Stress-activated protein kinase/Jun N-terminal kinase is required for interleukin (IL)-1-induced IL-6 and IL-8 gene expression in the human epidermal carcinoma cell line KB.

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Differential sensitivity of breast cancer cells to tumor necrosis factor-alpha: involvement of protein kinase C.

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21 January 2018

Identifiers

10.1074/JBC.M200468200

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

23 September 2017

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