Caspase inhibition activates HIV in latently infected cells. Role of tumor necrosis factor receptor 1 and CD95. (Q40749727)

23 September 2017

title

Caspase inhibition activates HIV in latently infected cells. Role of tumor necrosis factor receptor 1 and CD95. (English)

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23 September 2017

7

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23 September 2017

Sieghart Sopper

2

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Volker ter Meulen

8

object named as

Volker ter Meulen

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23 September 2017

Carsten Scheller

1

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23 September 2017

Peifeng Chen

3

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23 September 2017

Egbert Flory

4

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23 September 2017

Eleni Koutsilieri

5

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23 September 2017

Tomas Racek

6

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23 September 2017

Christian Jassoy

9

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23 September 2017

language of work or name

English

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publication date

19 February 2002

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Journal of Biological Chemistry

23 September 2017

published in

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23 September 2017

volume

277

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23 September 2017

issue

18

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23 September 2017

page(s)

15459-15464

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Signal transduction by tumor necrosis factor and its relatives

23 September 2017

cites work

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21 January 2018

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FADD, a novel death domain-containing protein, interacts with the death domain of Fas and initiates apoptosis

21 January 2018

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Protease activation during apoptosis: death by a thousand cuts?

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The CD95 (APO-1/Fas) receptor activates NF-kappaB independently of its cytotoxic function

21 January 2018

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Tumor necrosis factor alpha activates human immunodeficiency virus type 1 through induction of nuclear factor binding to the NF-kappa B sites in the long terminal repeat

21 January 2018

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Regulation of HIV-1 long terminal repeats by interaction of C/EBP(NF-IL6) and NF-kappaB/Rel transcription factors.

21 January 2018

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Induction of HIV-1 replication in latently infected CD4+ T cells using a combination of cytokines

21 January 2018

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Reduction of the HIV-1-infected T-cell reservoir by immune activation treatment is dose-dependent and restricted by the potency of antiretroviral drugs.

21 January 2018

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The number of HIV DNA-infected mononuclear cells is reduced under HAART plus recombinant IL-2. IRHAN Study Group

21 January 2018

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Latent reservoirs of HIV: obstacles to the eradication of virus

21 January 2018

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Tumor necrosis factor alpha induces expression of human immunodeficiency virus in a chronically infected T-cell clone

21 January 2018

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N-acetyl-leucinyl-leucinyl-norleucinal inhibits lipopolysaccharide-induced NF-kappaB activation and prevents TNF and IL-6 synthesis in vivo

21 January 2018

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Caspase-induced inactivation of the anti-apoptotic TRAF1 during Fas ligand-mediated apoptosis

21 January 2018

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Cleavage of the death domain kinase RIP by caspase-8 prompts TNF-induced apoptosis

21 January 2018

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TRAF1 is a substrate of caspases activated during tumor necrosis factor receptor-alpha-induced apoptosis

21 January 2018

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Caspase-dependent cleavage of signaling proteins during apoptosis. A turn-off mechanism for anti-apoptotic signals

21 January 2018

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Caspase-cleaved TRAF1 negatively regulates the antiapoptotic signals of TRAF2 during TNF-induced cell death

21 January 2018

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Characterization of a continuous T-cell line susceptible to the cytopathic effects of the acquired immunodeficiency syndrome (AIDS)-associated retrovirus

21 January 2018

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The inhibition of pro-apoptotic ICE-like proteases enhances HIV replication

21 January 2018

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Different Mitogen-activated Protein Kinase Signaling Pathways Cooperate to Regulate Tumor Necrosis Factor α Gene Expression in T Lymphocytes

21 January 2018

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HIV-1 LTR activity in human CD40-activated B lymphocytes is dependent on NF-kappaB

21 January 2018

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Effect of interleukin-2 on the pool of latently infected, resting CD4+ T cells in HIV-1-infected patients receiving highly active anti-retroviral therapy

21 January 2018

1 reference

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Effects of therapy with highly active anti-retroviral therapy (HAART) and IL-2 on CD4+ and CD8+ lymphocyte apoptosis in HIV+ patients

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M200763200

21 January 2018

Identifiers

DOI

10.1074/JBC.M200763200

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23 September 2017

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