p53 dependent apoptosis in glioma cell lines in response to hydrogen peroxide induced oxidative stress (Q40755110)

23 September 2017

title

p53 dependent apoptosis in glioma cell lines in response to hydrogen peroxide induced oxidative stress (English)

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23 September 2017

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ChEBI release 2020-05-28

Kamal Datta

1

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23 September 2017

Preeti Babbar

2

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23 September 2017

Tapasya Srivastava

3

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23 September 2017

Subrata Sinha

4

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23 September 2017

Parthaprasad Chattopadhyay

5

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23 September 2017

publication date

1 February 2002

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International Journal of Biochemistry & Cell Biology

23 September 2017

published in

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23 September 2017

volume

34

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23 September 2017

issue

2

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23 September 2017

page(s)

148-157

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Strategies of antioxidant defense

23 September 2017

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Oxidative stress as a mediator of apoptosis

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Hydrogen peroxide-induced apoptosis is CD95-independent, requires the release of mitochondria-derived reactive oxygen species and the activation of NF-kappaB.

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Differential effect of hydrogen peroxide and superoxide anion on apoptosis and proliferation of vascular smooth muscle cells.

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Methyl methanesulfonate and hydrogen peroxide differentially regulate p53 accumulation in hepatoblastoma cells

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Active oxygen species mediate the solar ultraviolet radiation-dependent increase in the tumour suppressor protein p53 in human skin fibroblasts.

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The induction of apoptosis in proliferating human fibroblasts by oxygen radicals is associated with a p53- and p21WAF1CIP1 induction

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Signaling pathways in reactive oxygen species-induced cardiomyocyte apoptosis

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p53 status and the efficacy of cancer therapy in vivo

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7 January 2021

p53 interacts with the DNA mismatch repair system to modulate the cytotoxicity and mutagenicity of hydrogen peroxide

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Role of reactive oxygen species and p53 in chromium(VI)-induced apoptosis

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A unique glucose-dependent apoptotic pathway induced by c-Myc

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Apoptosis provoked by the oxidative stress inducer menadione (Vitamin K(3)) is mediated by the Fas/Fas ligand system

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Nuclear factor-kappa B.

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7 January 2021

Bcl-2 down-regulates the activity of transcription factor NF-kappaB induced upon apoptosis

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7 January 2021

Bcl-2 suppresses apoptosis resulting from disruption of the NF-kappa B survival pathway

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The subpopulations and isolated cell types of freshly resected high grade human gliomas: their influence on the tumor's evolution in vivo and behavior and therapy in vitro

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Increase in external glutamate and NMDA receptor activation contribute to H2O2-induced neuronal apoptosis

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Identifiers

DOI

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23 September 2017

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