Depletion of intracellular calcium stores is toxic to SH-SY5Y neuronal cells (Q40762099)

23 September 2017

Depletion of intracellular calcium stores is toxic to SH-SY5Y neuronal cells (English)

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23 September 2017

Henry N Nguyen

1

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PubMed ID

11750901

23 September 2017

Chen Wang

series ordinal

2

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PubMed ID

11750901

23 September 2017

David C Perry

series ordinal

3

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23 September 2017

1 January 2002

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23 September 2017

8

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based on heuristic

inferred from page(s)

published in

Brain Research

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23 September 2017

924

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23 September 2017

2

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23 September 2017

159-166

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Inositol trisphosphate and calcium signalling

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Ca2+ entry following store depletion in SH-SY5Y neuroblastoma cells

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Ionized intracellular calcium concentration predicts excitotoxic neuronal death: observations with low-affinity fluorescent calcium indicators

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Effects of bipyridylium compounds on calcium release from triadic vesicles isolated from rabbit skeletal muscle

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Calcium in ischemic cell death

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Differential expression and regulation of ryanodine receptor and myo-inositol 1,4,5-trisphosphate receptor Ca2+ release channels in mammalian tissues and cell lines

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Alterations of Extracellular Calcium Elicit Selective Modes of Cell Death and Protease Activation in SH-SY5Y Human Neuroblastoma Cells

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1 reference

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The endoplasmic reticulum Ca2+ store: a view from the lumen

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Endoplasmicreticulum-induced signal transduction and gene expression

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Signal transduction from the endoplasmic reticulum to the cell nucleus

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Disturbances of the functioning of endoplasmic reticulum: a key mechanism underlying neuronal cell injury?

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Molecular and cellular physiology of intracellular calcium stores

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Distinct influx pathways, not calcium load, determine neuronal vulnerability to calcium neurotoxicity.

7 January 2021

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Calcium fluxes, calcium antagonists, and calcium-related pathology in brain ischemia, hypoglycemia, and spreading depression: a unifying hypothesis

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Is the cell death pathway triggered by the mitochondrion or the endoplasmic reticulum?

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Dantrolene. A review of its pharmacodynamic and pharmacokinetic properties and therapeutic use in malignant hyperthermia, the neuroleptic malignant syndrome and an update of its use in muscle spasticity

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Regulation of translational initiation during cellular responses to stress

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inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1016/S0006-8993(01)03229-2

1 reference

23 September 2017

1 reference

PubMed ID

11750901