A case of neuroendocrine oncogenic osteomalacia associated with a PHEX and fibroblast growth factor-23 expressing sinusidal malignant schwannoma. (Q40775606)

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English	A case of neuroendocrine oncogenic osteomalacia associated with a PHEX and fibroblast growth factor-23 expressing sinusidal malignant schwannoma.	scientific article

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scholarly article

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12 November 2019

A case of neuroendocrine oncogenic osteomalacia associated with a PHEX and fibroblast growth factor-23 expressing sinusidal malignant schwannoma (English)

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12 November 2019

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12 November 2019

author name string

M R John

1

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12 November 2019

H Wickert

2

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12 November 2019

K Zaar

3

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12 November 2019

K B Jonsson

4

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12 November 2019

A Grauer

5

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12 November 2019

P Ruppersberger

6

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12 November 2019

H Schmidt-Gayk

7

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12 November 2019

H Murer

8

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12 November 2019

R Ziegler

9

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publication date

1 October 2001

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12 November 2019

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12 November 2019

29

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393-402

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4

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12 November 2019

Molecular cloning and chromosomal mapping of DNA rearranged with the parathyroid hormone gene in a parathyroid adenoma

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Aberrant regulation of ras proteins in malignant tumour cells from type 1 neurofibromatosis patients

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Circulating levels of midregional parathyroid hormone-related protein in hypercalcaemia of malignancy

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Brief report: inhibition of renal phosphate transport by a tumor product in a patient with oncogenic osteomalacia

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Hypophosphatemia induced in mice by transplantation of a tumor-derived cell line from a patient with oncogenic rickets

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Malignant peripheral nerve sheath tumors. A clinicopathologic study of 120 cases

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Autosomal dominant hypophosphatemic rickets is linked to chromosome 12p13.

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Genomic organization of the human PEX gene mutated in X-linked dominant hypophosphatemic rickets.

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Immunochemiluminometric assay with two monoclonal antibodies against the N-terminal sequence of human parathyroid hormone

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Expression and cloning of the human X-linked hypophosphatemia gene cDNA

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Vitamin D resistant osteomalacia

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7 January 2021

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Phosphatonin--a new phosphaturetic hormone? (lessons from tumour-induced osteomalacia and X-linked hypophosphataemia)

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7 January 2021

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Tumor-induced osteomalacia and the regulation of phosphate homeostasis

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Direct demonstration of a humorally-mediated inhibition of renal phosphate transport in the Hyp mouse

1 reference

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Cloning of human PEX cDNA. Expression, subcellular localization, and endopeptidase activity

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Cytogenetic characterization of peripheral nerve sheath tumours: a report of the CHAMP study group

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Hemangiopericytoma-induced osteomalacia: tumor transplantation in nude mice causes hypophosphatemia and tumor extracts inhibit renal 25-hydroxyvitamin D 1-hydroxylase activity

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Characteristics of tumor cell bioactivity in oncogenic osteomalacia

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Coordinated maturational regulation of PHEX and renal phosphate transport inhibitory activity: evidence for the pathophysiological role of PHEX in X-linked hypophosphatemia

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Parathyroid hormone-dependent degradation of type II Na+/Pi cotransporters

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Multiple cytogenetic aberrations in neurofibrosarcomas complicating neurofibromatosis

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Candidate 56 and 58 kDa protein(s) responsible for mediating the renal defects in oncogenic hypophosphatemic osteomalacia

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Parathyroid hormone-like bioactivity in tumours from patients with oncogenic osteomalacia

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A neuroendocrine cause of oncogenic osteomalacia

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Pex gene deletions in Gy and Hyp mice provide mouse models for X-linked hypophosphatemia.

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Phosphaturic mesenchymal tumors. A polymorphous group causing osteomalacia or rickets

1 reference

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Autosomal dominant hypophosphataemic rickets is associated with mutations in FGF23

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The Autosomal Dominant Hypophosphatemic Rickets (ADHR) Gene Is a Secreted Polypeptide Overexpressed by Tumors that Cause Phosphate Wasting

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Identification of a novel fibroblast growth factor, FGF-23, preferentially expressed in the ventrolateral thalamic nucleus of the brain

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Identifiers

10.1016/S8756-3282(01)00586-5

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12 November 2019

1 reference

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12 November 2019

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