A mitogenic signal triggered at an early stage of vaccinia virus infection: implication of MEK/ERK and protein kinase A in virus multiplication. (Q40791679)

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English	A mitogenic signal triggered at an early stage of vaccinia virus infection: implication of MEK/ERK and protein kinase A in virus multiplication.	scientific article

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9 November 2019

A mitogenic signal triggered at an early stage of vaccinia virus infection: implication of MEK/ERK and protein kinase A in virus multiplication (English)

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9 November 2019

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vaccinia virus

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9 November 2019

Anderson Assunção Andrade

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9 November 2019

Ricardo Gazzinelli

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9 November 2019

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J C de Magalhães

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9 November 2019

P N Silva

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9 November 2019

L P Sousa

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9 November 2019

C Ropert

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9 November 2019

P C Ferreira

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9 November 2019

C A Bonjardim

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9 November 2019

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17 July 2001

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9 November 2019

Journal of Biological Chemistry

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9 November 2019

276

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9 November 2019

38353-38360

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9 November 2019

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9 November 2019

Vaccinia virus-infected cells release a novel polypeptide functionally related to transforming and epidermal growth factors

1 reference

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21 January 2018

Cell proliferative response to vaccinia virus is mediated by VGF.

1 reference

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21 January 2018

Vaccinia virus 19-kilodalton protein: relationship to several mammalian proteins, including two growth factors

1 reference

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21 January 2018

Vaccinia virus encodes a polypeptide homologous to epidermal growth factor and transforming growth factor

1 reference

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The genome of cowpox virus contains a gene related to those encoding the epidermal growth factor, transforming growth factor alpha and vaccinia growth factor

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Induction of c-fos gene and protein by growth factors precedes activation of c-myc

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Continuous c-fos expression precedes programmed cell death in vivo

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Functional role for c-myc in mitogenic response to platelet-derived growth factor

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Transcription factor p91 interacts with the epidermal growth factor receptor and mediates activation of the c-fos gene promoter

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Activation of Proto-Oncogenes: an Immediate Early Event in Human Cytomegalovirus Infection

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Herpes simplex virus type 1 infection stimulates p38/c-Jun N-terminal mitogen-activated protein kinase pathways and activates transcription factor AP-1.

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Activation of the transforming potential of the human fos proto-oncogene requires message stabilization and results in increased amounts of partially modified fos protein

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Morphological and molecular characterization of the poxvirus BeAn 58058.

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Genomic sequencing

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Biological activities of a human amniotic membrane interferon

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A mutant cell line partially responsive to both IFN-alpha and IFN-gamma

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Signalling by receptor tyrosine kinases

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Transcriptional control by protein phosphorylation: signal transmission from the cell surface to the nucleus

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PD 098059 is a specific inhibitor of the activation of mitogen-activated protein kinase kinase in vitro and in vivo

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Role of mitogen-activated protein kinase cascades in mediating lipopolysaccharide-stimulated induction of cyclooxygenase-2 and IL-1 beta in RAW264 macrophages

1 reference

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A role for protein kinase cepsilon in the inhibitory effect of epidermal growth factor on calcium-stimulated chloride secretion in human colonic epithelial cells

1 reference

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21 January 2018

Entry of the two infectious forms of vaccinia virus at the plasma membane is signaling-dependent for the IMV but not the EEV.

1 reference

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21 January 2018

Epidermal growth factor induces phosphorylation of extracellular signal-regulated kinase 2 via multiple pathways

1 reference

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Protein kinase C--a question of specificity

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Phosphorylation of transcription factor p62TCF by MAP kinase stimulates ternary complex formation at c-fos promoter

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Rsk-2 activity is necessary for epidermal growth factor-induced phosphorylation of CREB protein and transcription of c-fos gene

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ERK activation induces phosphorylation of Elk-1 at multiple S/T-P motifs to high stoichiometry

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The low proliferation rates of human amniotic cells are neither associated to deregulated proto-oncogenes' expression nor to the effect of IFNα2

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10.1074/JBC.M100183200

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