Cleavage of Bax is mediated by caspase-dependent or -independent calpain activation in dopaminergic neuronal cells: protective role of Bcl-2. (Q40798203)

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English	Cleavage of Bax is mediated by caspase-dependent or -independent calpain activation in dopaminergic neuronal cells: protective role of Bcl-2.	scientific article

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scholarly article

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Europe PubMed Central

23 September 2017

Cleavage of Bax is mediated by caspase-dependent or -independent calpain activation in dopaminergic neuronal cells: protective role of Bcl-2. (English)

1 reference

Europe PubMed Central

23 September 2017

1 reference

based on heuristic

inferred from title

4

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Europe PubMed Central

23 September 2017

author name string

Choi WS

1

1 reference

Europe PubMed Central

23 September 2017

Lee EH

2

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Europe PubMed Central

23 September 2017

Chung CW

3

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Europe PubMed Central

23 September 2017

Jin BK

5

1 reference

Europe PubMed Central

23 September 2017

Kim SU

6

1 reference

Europe PubMed Central

23 September 2017

Oh TH

7

1 reference

Europe PubMed Central

23 September 2017

Saido TC

8

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Europe PubMed Central

23 September 2017

Oh YJ

9

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Europe PubMed Central

23 September 2017

language of work or name

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publication date

1 June 2001

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Europe PubMed Central

23 September 2017

Journal of Neurochemistry

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Europe PubMed Central

23 September 2017

77

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Europe PubMed Central

23 September 2017

1531-1541

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Europe PubMed Central

23 September 2017

6

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Europe PubMed Central

23 September 2017

Inhibition of Bax channel-forming activity by Bcl-2

1 reference

https://api.crossref.org/works/10.1046%2FJ.1471-4159.2001.00368.X

21 January 2018

Subcellular compartmentalization of calcium-dependent and calcium-independent neutral proteases in brain

1 reference

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Inhibition of ICE family proteases by baculovirus antiapoptotic protein p35.

1 reference

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Immortalization of embryonic mesencephalic dopaminergic neurons by somatic cell fusion

1 reference

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Specific modulation of dopamine expression in neuronal hybrid cells by primary cells from different brain regions

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https://api.crossref.org/works/10.1046%2FJ.1471-4159.2001.00368.X

21 January 2018

Direct cleavage by the calcium-activated protease calpain can lead to inactivation of caspases

1 reference

https://api.crossref.org/works/10.1046%2FJ.1471-4159.2001.00368.X

21 January 2018

Annexin V-affinity assay: a review on an apoptosis detection system based on phosphatidylserine exposure

1 reference

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21 January 2018

Alphaviruses induce apoptosis in Bcl-2-overexpressing cells: evidence for a caspase-mediated, proteolytic inactivation of Bcl-2.

1 reference

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21 January 2018

Enforced dimerization of BAX results in its translocation, mitochondrial dysfunction and apoptosis

1 reference

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21 January 2018

Glial-derived neurotrophic factor (GDNF) induced morphological differentiation of an immortalized monoclonal hybrid dopaminergic cell line of mesencephalic neuronal origin.

1 reference

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21 January 2018

Programmed cell death and Bcl-2 protection in very low oxygen.

1 reference

https://api.crossref.org/works/10.1046%2FJ.1471-4159.2001.00368.X

21 January 2018

Bax directly induces release of cytochrome c from isolated mitochondria

1 reference

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21 January 2018

Mechanisms of calpain proteolysis following traumatic brain injury: implications for pathology and therapy: implications for pathology and therapy: a review and update

1 reference

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21 January 2018

Expression of bcl-2 inhibits necrotic neural cell death.

1 reference

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21 January 2018

Sequential cleavage of poly(ADP-ribose)polymerase and appearance of a small Bax-immunoreactive protein are blocked by Bcl-X(L) and caspase inhibitors during staurosporine-induced dopaminergic neuronal apoptosis

1 reference

https://api.crossref.org/works/10.1046%2FJ.1471-4159.2001.00368.X

21 January 2018

Protease activation during apoptosis: death by a thousand cuts?

1 reference

https://api.crossref.org/works/10.1046%2FJ.1471-4159.2001.00368.X

21 January 2018

Increased M-calpain expression in the mesencephalon of patients with Parkinson's disease but not in other neurodegenerative disorders involving the mesencephalon: a role in nerve cell death?

1 reference

https://api.crossref.org/works/10.1046%2FJ.1471-4159.2001.00368.X

21 January 2018

Overexpression of HA-Bax but not Bcl-2 or Bcl-XL attenuates 6-hydroxydopamine-induced neuronal apoptosis

1 reference

https://api.crossref.org/works/10.1046%2FJ.1471-4159.2001.00368.X

21 January 2018

Overexpression of Bcl-2 attenuates MPP+, but not 6-ODHA, induced cell death in a dopaminergic neuronal cell line

1 reference

https://api.crossref.org/works/10.1046%2FJ.1471-4159.2001.00368.X

21 January 2018

Regions outside of the Bcl-2 homology domains, BH1 and BH2 protect a dopaminergic neuronal cell line from staurosporine-induced cell death

1 reference

https://api.crossref.org/works/10.1046%2FJ.1471-4159.2001.00368.X

21 January 2018

Implication of calpain in caspase activation during B cell clonal deletion

1 reference

https://api.crossref.org/works/10.1046%2FJ.1471-4159.2001.00368.X

21 January 2018

Widespread activation of calcium-activated neutral proteinase (calpain) in the brain in Alzheimer disease: a potential molecular basis for neuronal degeneration

1 reference

https://api.crossref.org/works/10.1046%2FJ.1471-4159.2001.00368.X

21 January 2018

Comparison of the ion channel characteristics of proapoptotic BAX and antiapoptotic BCL-2.

1 reference

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21 January 2018

Inhibition of PC12 cell redox activity is a specific, early indicator of the mechanism of beta-amyloid-mediated cell death

1 reference

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21 January 2018

A putative mechanism of demyelination in multiple sclerosis by a proteolytic enzyme, calpain

1 reference

https://api.crossref.org/works/10.1046%2FJ.1471-4159.2001.00368.X

21 January 2018

Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC

1 reference

https://api.crossref.org/works/10.1046%2FJ.1471-4159.2001.00368.X

21 January 2018

Purification from Synaptosomal Plasma Membranes of Calpain I, a Thiol Protease Activated by Micromolar Calcium Concentrations

1 reference

https://api.crossref.org/works/10.1046%2FJ.1471-4159.2001.00368.X

21 January 2018

Structure and physiological function of calpains

1 reference

https://api.crossref.org/works/10.1046%2FJ.1471-4159.2001.00368.X

21 January 2018

Expression of Alzheimer's disease-associated presenilin-1 is controlled by proteolytic degradation and complex formation

1 reference

https://api.crossref.org/works/10.1046%2FJ.1471-4159.2001.00368.X

21 January 2018

Calpain inhibitors as therapeutic agents in nerve and muscle degeneration

1 reference

https://api.crossref.org/works/10.1046%2FJ.1471-4159.2001.00368.X

21 January 2018

Apoptosis in the pathogenesis and treatment of disease

1 reference

https://api.crossref.org/works/10.1046%2FJ.1471-4159.2001.00368.X

21 January 2018

Caspases: enemies within

1 reference

https://api.crossref.org/works/10.1046%2FJ.1471-4159.2001.00368.X

21 January 2018

A novel assay for apoptosis. Flow cytometric detection of phosphatidylserine expression on early apoptotic cells using fluorescein labelled Annexin V

1 reference

https://api.crossref.org/works/10.1046%2FJ.1471-4159.2001.00368.X

21 January 2018

Calpain and caspase: can you tell the difference?

1 reference

https://api.crossref.org/works/10.1046%2FJ.1471-4159.2001.00368.X

21 January 2018

Caspase-mediated fragmentation of calpain inhibitor protein calpastatin during apoptosis

1 reference

https://api.crossref.org/works/10.1046%2FJ.1471-4159.2001.00368.X

21 January 2018

Movement of Bax from the cytosol to mitochondria during apoptosis

1 reference

https://api.crossref.org/works/10.1046%2FJ.1471-4159.2001.00368.X

21 January 2018

Cleavage of Bax enhances its cell death function

1 reference

https://api.crossref.org/works/10.1046%2FJ.1471-4159.2001.00368.X

21 January 2018

Calpain inhibitor I increases beta-amyloid peptide production by inhibiting the degradation of the substrate of gamma-secretase. Evidence that substrate availability limits beta-amyloid peptide production

1 reference

https://api.crossref.org/works/10.1046%2FJ.1471-4159.2001.00368.X

21 January 2018

Identifiers

10.1046/J.1471-4159.2001.00368.X

1 reference

Europe PubMed Central

23 September 2017

1 reference

Europe PubMed Central

23 September 2017

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