Notch(ic)-ER chimeras display hormone-dependent transformation, nuclear accumulation, phosphorylation and CBF1 activation. (Q40860023)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10951584%20AND%20SRC:MED&resulttype=core&format=json

9 November 2019

title

Notch(ic)-ER chimeras display hormone-dependent transformation, nuclear accumulation, phosphorylation and CBF1 activation (English)

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9 November 2019

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1

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9 November 2019

author name string

A J Capobianco

series ordinal

2

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9 November 2019

language of work or name

English

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publication date

1 August 2000

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9 November 2019

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9 November 2019

volume

19

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9 November 2019

page(s)

3914-3924

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9 November 2019

issue

34

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https://scigraph.springernature.com/pub.10.1038/sj.onc.1203719

9 November 2019

exact match

0 references

1 reference

https://api.crossref.org/works/10.1038%2FSJ.ONC.1203719

Notch signaling: cell fate control and signal integration in development

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Functional analysis of the TAN-1 gene, a human homolog of Drosophila notch

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A conditional mutant of vRel containing sequences from the human estrogen receptor

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Neoplastic transformation by truncated alleles of human NOTCH1/TAN1 and NOTCH2.

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MYC abrogates p53-mediated cell cycle arrest in N-(phosphonacetyl)-L-aspartate-treated cells, permitting CAD gene amplification

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The link between integration and expression of human papillomavirus type 16 genomes and cellular changes in the evolution of cervical intraepithelial neoplastic lesions

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Involvement of Notch1 in the development of mouse mammary tumors

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TAN-1, the human homolog of the Drosophila Notch gene, is broken by chromosomal translocations in T lymphoblastic neoplasms

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Complex cellular and subcellular regulation of notch expression during embryonic and imaginal development of Drosophila: implications for notch function

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Transient excess of MYC activity can elicit genomic instability and tumorigenesis

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The suppressor of hairless protein participates in notch receptor signaling.

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An activated Notch receptor blocks cell-fate commitment in the developing Drosophila eye.

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The choice of cell fate in the epidermis of Drosophila.

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Truncated mammalian Notch1 activates CBF1/RBPJk-repressed genes by a mechanism resembling that of Epstein-Barr virus EBNA2

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Signalling downstream of activated mammalian Notch

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Neoplastic transformation by Notch requires nuclear localization

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Expression of an activated Notch-related int-3 transgene interferes with cell differentiation and induces neoplastic transformation in mammary and salivary glands

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A histone deacetylase corepressor complex regulates the Notch signal transduction pathway

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Structure and distribution of the Notch protein in developing Drosophila.

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Ligand-induced cleavage and regulation of nuclear entry of Notch in Drosophila melanogaster embryos

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Signal transduction by activated mNotch: importance of proteolytic processing and its regulation by the extracellular domain.

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Hormone-regulatable neoplastic transformation induced by a Jun-estrogen receptor chimera

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Indirect evidence for Delta-dependent intracellular processing of notch in Drosophila embryos

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Antineurogenic phenotypes induced by truncated Notch proteins indicate a role in signal transduction and may point to a novel function for Notch in nuclei

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Arbiter of differentiation and death: Notch signaling meets apoptosis

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NF-kappaB2 is a putative target gene of activated Notch-1 via RBP-Jkappa

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Exclusive development of T cell neoplasms in mice transplanted with bone marrow expressing activated Notch alleles

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Specific truncations of Drosophila Notch define dominant activated and dominant negative forms of the receptor.

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The activation domain of a hormone inducible HTLV-1 Rex protein determines colocalization with the nuclear pore.

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Conditional transformation of cells and rapid activation of the mitogen-activated protein kinase cascade by an estradiol-dependent human raf-1 protein kinase

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Notch-1 signalling requires ligand-induced proteolytic release of intracellular domain

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Nuclear access and action of notch in vivo

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Intrinsic activity of the Lin-12 and Notch intracellular domains in vivo

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Hormone-dependent transcriptional regulation and cellular transformation by Fos-steroid receptor fusion proteins

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Physical interaction between a novel domain of the receptor Notch and the transcription factor RBP-J kappa/Su(H)

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CDC25A phosphatase is a target of E2F and is required for efficient E2F-induced S phase

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Alterations in Notch signaling in neoplastic lesions of the human cervix

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Chimaeras of Myc oncoprotein and steroid receptors cause hormone-dependent transformation of cells

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Frequent provirus insertional mutagenesis of Notch1 in thymomas of MMTVD/myc transgenic mice suggests a collaboration of c-myc and Notch1 for oncogenesis

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Involvement of Notch-1 in mammalian retinal neurogenesis: association of Notch-1 activity with both immature and terminally differentiated cells

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7 January 2021

Phosphorylation of the human estrogen receptor on tyrosine 537 in vivo and by src family tyrosine kinases in vitro

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Neoplastic transformation by Notch is independent of transcriptional activation by RBP-J signalling

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Characterization of the CBF2 binding site within the Epstein-Barr virus latency C promoter and its role in modulating EBNA2-mediated transactivation.

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Hormone-conditional transformation by fusion proteins of c-Abl and its transforming variants

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Mouse mammary tumor gene int-3: a member of the notch gene family transforms mammary epithelial cells

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Human homologs of a Drosophila Enhancer of split gene product define a novel family of nuclear proteins

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Identifiers

DOI

10.1038/SJ.ONC.1203719

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10951584%20AND%20SRC:MED&resulttype=core&format=json

Dimensions Publication ID

9 November 2019

0 references

1 reference