STAT3 mediates IL-6-induced neuroendocrine differentiation in prostate cancer cells . (Q40905638)

24 September 2017

title

STAT3 mediates IL-6-induced neuroendocrine differentiation in prostate cancer cells . (English)

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24 September 2017

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Spiotto MT

1

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24 September 2017

Chung TD

series ordinal

2

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24 September 2017

publication date

1 February 2000

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24 September 2017

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24 September 2017

volume

42

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24 September 2017

issue

3

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24 September 2017

page(s)

186-195

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24 September 2017

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Etk/Bmx, a tyrosine kinase with a pleckstrin-homology domain, is an effector of phosphatidylinositol 3'-kinase and is involved in interleukin 6-induced neuroendocrine differentiation of prostate cancer cells

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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Interleukin-6 (IL-6) functions as an in vitro autocrine growth factor in renal cell carcinomas

21 January 2018

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Characterization of the role of IL-6 in the progression of prostate cancer

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Differentiation and growth arrest signals are generated through the cytoplasmic region of gp130 that is essential for Stat3 activation

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Neuroendocrine differentiation in prostate cancer: enhanced prediction of progression after radical prostatectomy

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A major role for the protein tyrosine kinase JAK1 in the JAK/STAT signal transduction pathway in response to interleukin-6.

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Activation of gp 130 by IL-6/soluble IL-6 receptor induces neuronal differentiation

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Relationship between cAMP induced inhibition of human meningioma cell proliferation and autocrine secretion of interleukin-6

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A central role for Stat3 in IL-6-induced regulation of growth and differentiation in M1 leukemia cells

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Two distinct and independent sites on IL-6 trigger gp 130 dimer formation and signalling

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Mechanism of retinoblastoma gene inactivation in the spectrum of neuroendocrine lung tumors

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Identifiers

DOI

10.1002/(SICI)1097-0045(20000215)42:3<186::AID-PROS4>3.0.CO;2-E

1 reference

24 September 2017

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