Changes in the balance of phosphoinositide 3-kinase/protein kinase B (Akt) and the mitogen-activated protein kinases (ERK/p38MAPK) determine a phenotype of visceral and vascular smooth muscle cells (Q40953884)

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scientific article published on May 1999

Language	Label	Description	Also known as
English	Changes in the balance of phosphoinositide 3-kinase/protein kinase B (Akt) and the mitogen-activated protein kinases (ERK/p38MAPK) determine a phenotype of visceral and vascular smooth muscle cells	scientific article published on May 1999

Statements

scholarly article

1 reference

Europe PubMed Central

25 September 2017

Changes in the balance of phosphoinositide 3-kinase/protein kinase B (Akt) and the mitogen-activated protein kinases (ERK/p38MAPK) determine a phenotype of visceral and vascular smooth muscle cells (English)

1 reference

Europe PubMed Central

25 September 2017

author name string

K Hayashi

1

1 reference

Europe PubMed Central

25 September 2017

M Takahashi

2

1 reference

Europe PubMed Central

25 September 2017

K Kimura

3

1 reference

Europe PubMed Central

25 September 2017

W Nishida

4

1 reference

Europe PubMed Central

25 September 2017

H Saga

5

1 reference

Europe PubMed Central

25 September 2017

K Sobue

6

1 reference

Europe PubMed Central

25 September 2017

language of work or name

0 references

publication date

1 May 1999

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Europe PubMed Central

25 September 2017

number of pages

14

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based on heuristic

inferred from page(s)

Journal of Cell Biology

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Europe PubMed Central

25 September 2017

145

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Europe PubMed Central

25 September 2017

4

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Europe PubMed Central

25 September 2017

727-740

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Europe PubMed Central

25 September 2017

The search for physiological substrates of MAP and SAP kinases in mammalian cells

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2133182

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A serum response factor-dependent transcriptional regulatory program identifies distinct smooth muscle cell sublineages

1 reference

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Tenascin-C, proliferation and subendothelial fibronectin in progressive pulmonary vascular disease.

1 reference

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Expression of the smooth muscle myosin heavy chain gene is regulated by a negative-acting GC-rich element located between two positive-acting serum response factor-binding elements

1 reference

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MKK3- and MKK6-regulated gene expression is mediated by the p38 mitogen-activated protein kinase signal transduction pathway

1 reference

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Acute hypertension activates mitogen-activated protein kinases in arterial wall

1 reference

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Isolation of gene markers of differentiated and proliferating vascular smooth muscle cells.

1 reference

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The pathogenesis of atherosclerosis: a perspective for the 1990s

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Insulin-like growth factor-I and platelet-derived growth factor-BB induce directed migration of human arterial smooth muscle cells via signaling pathways that are distinct from those of proliferation

1 reference

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Transformation of mammalian cells by constitutively active MAP kinase kinase

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Identification of functional promoter elements in the rabbit smooth muscle myosin heavy chain gene.

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MAPKs: new JNK expands the group

1 reference

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Regulation of differentiation of vascular smooth muscle cells

1 reference

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Expression of high and low molecular weight caldesmons during phenotypic modulation of smooth muscle cells

1 reference

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Insulin-stimulated microtubule-associated protein kinase is phosphorylated on tyrosine and threonine in vivo

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Metavinculin and vinculin from mammalian smooth muscle: bulk isolation and characterization

1 reference

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Caldesmon, a novel regulatory protein in smooth muscle and nonmuscle actomyosin systems

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Calponin and SM 22 isoforms in avian and mammalian smooth muscle. Absence of phosphorylation in vivo

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Genomic structure of the human caldesmon gene

1 reference

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The smooth muscle cell in culture

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Sounding the alarm: protein kinase cascades activated by stress and inflammation

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Signal-transducing protein phosphorylation cascades mediated by Ras/Rho proteins in the mammalian cell: the potential for multiplex signalling

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Transcriptional regulation of the chicken caldesmon gene. Activation of gizzard-type caldesmon promoter requires a CArG box-like motif

1 reference

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Mechanism of activation and function of protein kinase B.

1 reference

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Platelet-derived growth factor activates a mammalian Ste20 coupled mitogen-activated protein kinase in airway smooth muscle

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Ras-dependent induction of cellular responses by constitutively active phosphatidylinositol-3 kinase

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Variables controlling the secretion of a somatomedin-like peptide by cultured porcine smooth muscle cells

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Identification of two types of smooth muscle myosin heavy chain isoforms by cDNA cloning and immunoblot analysis.

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Developmentally regulated expression of vascular smooth muscle myosin heavy chain isoforms.

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Role of the mitogen-activated protein kinases in the expression of the kinin B1 receptors induced by tissue injury

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Smooth muscle cell phenotype-dependent transcriptional regulation of the alpha1 integrin gene.

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1 reference

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Structure and expression of a smooth muscle cell-specific gene, SM22 alpha.

1 reference

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1 reference

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Molecular mechanism of phenotypic modulation of smooth muscle cells

1 reference

https://pubmed.ncbi.nlm.nih.gov/10330402

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Regulation of insulin-like growth factor-I gene expression by growth factors in cultured vascular smooth muscle cells

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1 reference

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Caldesmon: A common actin-linked regulatory protein in the smooth muscle and nonmuscle contractile system

1 reference

https://pubmed.ncbi.nlm.nih.gov/10330402

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Regulation of insulin-like growth factor I messenger RNA levels in vascular smooth muscle cells

1 reference

https://pubmed.ncbi.nlm.nih.gov/10330402

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Activation of mitogen-activated protein kinases (ERK/JNK) and AP-1 transcription factor in rat carotid arteries after balloon injury

1 reference

https://pubmed.ncbi.nlm.nih.gov/10330402

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Autocrine FGF signaling is required for vascular smooth muscle cell survival in vitro

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https://pubmed.ncbi.nlm.nih.gov/10330402

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Differentiated phenotype of smooth muscle cells depends on signaling pathways through insulin-like growth factors and phosphatidylinositol 3-kinase

1 reference

https://pubmed.ncbi.nlm.nih.gov/10330402

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Activation of MAP kinase in vivo follows balloon overstretch injury of porcine coronary and carotid arteries

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https://pubmed.ncbi.nlm.nih.gov/10330402

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Identifiers

10.1083/JCB.145.4.727

1 reference

Europe PubMed Central

25 September 2017

1 reference

Europe PubMed Central

25 September 2017

1 reference

Europe PubMed Central

25 September 2017

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