Die and let live: eliminating dangerous lymphocytes (Q40984215)

25 September 2017

title

Die and let live: eliminating dangerous lymphocytes (English)

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25 September 2017

1

Abbas AK

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25 September 2017

language of work or name

English

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publication date

1 March 1996

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25 September 2017

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25 September 2017

volume

84

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25 September 2017

issue

5

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25 September 2017

page(s)

655-657

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A role for CD95 ligand in preventing graft rejection

25 September 2017

cites work

1 reference

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Contenders in FasL/TNF death signaling

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T cell deletion in high antigen dose therapy of autoimmune encephalomyelitis

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7 January 2021

Dominant interfering Fas gene mutations impair apoptosis in a human autoimmune lymphoproliferative syndrome

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7 January 2021

Fas Ligand-Induced Apoptosis as a Mechanism of Immune Privilege

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7 January 2021

Anatomy of autoantibody production: Dominant localization of antibody-producing cells to T cell zones in fas-deficient mice

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7 January 2021

Interleukin-2 programs mouse alpha beta T lymphocytes for apoptosis

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7 January 2021

Fas and Fas ligand: a death factor and its receptor.

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7 January 2021

Fas and Fas ligand: lpr and gld mutations

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7 January 2021

CD95 (Fas)-dependent elimination of self-reactive B cells upon interaction with CD4+T cells

1 reference

https://api.crossref.org/works/10.1016%2FS0092-8674%2800%2981042-9

7 January 2021

Mutations in Fas associated with human lymphoproliferative syndrome and autoimmunity

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https://api.crossref.org/works/10.1016%2FS0092-8674%2800%2981042-9

7 January 2021

The fas antigen is involved in peripheral but not thymic deletion of T lymphocytes in T cell receptor transgenic mice

1 reference

https://api.crossref.org/works/10.1016%2FS0092-8674%2800%2981042-9

7 January 2021

Molecular cloning and expression of the Fas ligand, a novel member of the tumor necrosis factor family

1 reference

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7 January 2021

Interleukin-2 receptor α chain regulates the size and content of the peripheral lymphoid compartment

1 reference

https://api.crossref.org/works/10.1016%2FS0092-8674%2800%2981042-9

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10.1016/S0092-8674(00)81042-9

Identifiers

DOI

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25 September 2017

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