Protein kinases C-gamma and -delta are involved in insulin-like growth factor I-induced migration of colonic epithelial cells (Q40985413)

25 September 2017

Protein kinases C-gamma and -delta are involved in insulin-like growth factor I-induced migration of colonic epithelial cells (English)

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25 September 2017

André F

1

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25 September 2017

Rigot V

2

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25 September 2017

Remacle-Bonnet M

3

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25 September 2017

Luis J

4

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25 September 2017

Pommier G

5

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25 September 2017

Marvaldi J

6

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25 September 2017

language of work or name

English

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publication date

1 January 1999

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25 September 2017

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25 September 2017

116

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25 September 2017

64-77

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25 September 2017

1

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Epithelial migration in the colon: filling in the gaps

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7 January 2021

based on heuristic

Role of extracellular matrix on colonic cancer cell migration and proliferation

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Interleukin 2 modulates intestinal epithelial cell function in vitro

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Cytokine modulation of intestinal epithelial cell restitution: central role of transforming growth factor beta

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Epidermal growth factor receptor-stimulated intestinal epithelial cell migration requires phospholipase C activity

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Hepatocyte growth factor/scatter factor modulates intestinal epithelial cell proliferation and migration

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Hepatocyte growth factor/scatter factor effects on epithelia. Regulation of intercellular junctions in transformed and nontransformed cell lines, basolateral polarization of c-met receptor in transformed and natural intestinal epithelia, and inducti

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Molecular and cellular aspects of the insulin-like growth factor I receptor

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Insulin-like Growth Factors as Regulators of Cell Motility Signaling Mechanisms

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Treatment with IGF-I peptides improves function of the remnant gut following small bowel resection in rats

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A Factor from a Transformed Cell Line That Affects Cell Migration

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Analysis of serum insulin-like growth factor binding proteins using western blotting: use of the method for titration of the binding proteins and competitive binding studies

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Cell polarity of the insulin-like growth factor system in human intestinal epithelial cells. Unique apical sorting of insulin-like growth factor binding protein-6 in differentiated human colon cancer cells

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Production of insulin-like growth factor II (IGF-II) and different forms of IGF-binding proteins by HT-29 human colon carcinoma cell line

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MAP kinase mediates epidermal growth factor- and phorbol ester-induced prostacyclin formation in cardiomyocytes.

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des-(1-3)-IGF-I, an insulin-like growth factor analog used to mimic a potential IGF-II autocrine loop, promotes the differentiation of human colon-carcinoma cells

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Expression of type I, but not type II insulin-like growth factor receptor on both undifferentiated and differentiated HT29 human colon carcinoma cell line

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Insulin-like growth factor-I and platelet-derived growth factor-BB induce directed migration of human arterial smooth muscle cells via signaling pathways that are distinct from those of proliferation

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Classification of the insulin-like growth factor binding proteins into three distinct categories according to their binding specificities

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Receptor tyrosine kinase signaling required for integrin alpha v beta 5-directed cell motility but not adhesion on vitronectin

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Calphostin C (UCN-1028C), a novel microbial compound, is a highly potent and specific inhibitor of protein kinase C

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The development of Na(+)-dependent glucose transport during differentiation of an intestinal epithelial cell clone is regulated by protein kinase C

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Protein kinase C isozymes and substrates

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Growth hormone and phorbol esters require specific protein kinase C isoforms to activate mitogen-activated protein kinases in 3T3-F442A cells

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Regulating integrin-mediated adhesion: one more function for PI 3-kinase?

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Requirement for phosphoinositide 3-OH kinase in growth hormone signalling to the mitogen-activated protein kinase and p70s6k pathways

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Insulin-like Growth Factor 1 Inhibits Apoptosis Using the Phosphatidylinositol 3′-Kinase and Mitogen-activated Protein Kinase Pathways

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Transforming growth factor-beta-induced cell growth inhibition in human breast cancer cells is mediated through insulin-like growth factor-binding protein-3 action.

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Integrins: versatility, modulation, and signaling in cell adhesion

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Signal transduction from the extracellular matrix

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Association of insulin receptor substrate-1 with integrins

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The roles of integrins and extracellular matrix proteins in the insulin-like growth factor I-stimulated chemotaxis of human breast cancer cells

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1 reference

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Ligand occupancy of the alpha-V-beta3 integrin is necessary for smooth muscle cells to migrate in response to insulin-like growth factor

7 January 2021

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Insulinlike growth factors and binding proteins in colon cancer

7 January 2021

1 reference

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inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1016/S0016-5085(99)70230-1

1 reference

25 September 2017

1 reference