Doxorubicin-induced apoptosis in human T-cell leukemia is mediated by caspase-3 activation in a Fas-independent way. (Q41074621)

26 September 2017

title

Doxorubicin-induced apoptosis in human T-cell leukemia is mediated by caspase-3 activation in a Fas-independent way. (English)

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26 September 2017

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Gamen S

1

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26 September 2017

Anel A

2

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26 September 2017

Lasierra P

3

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26 September 2017

Alava MA

4

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26 September 2017

Martinez-Lorenzo MJ

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26 September 2017

Piñeiro A

6

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26 September 2017

Naval J

7

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26 September 2017

language of work or name

English

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publication date

1 November 1997

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26 September 2017

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26 September 2017

volume

417

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26 September 2017

issue

3

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26 September 2017

page(s)

360-364

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ICE family proteases: mediators of all apoptotic cell death?

26 September 2017

cites work

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https://api.crossref.org/works/10.1016%2FS0014-5793%2897%2901282-9

Release of preformed Fas ligand in soluble form is the major factor for activation-induced death of Jurkat T cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2897%2901282-9

CPP32 inhibition prevents Fas-induced ceramide generation and apoptosis in human cells.

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2897%2901282-9

FADD, a novel death domain-containing protein, interacts with the death domain of Fas and initiates apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2897%2901282-9

In vitro activation of CPP32 and Mch3 by Mch4, a novel human apoptotic cysteine protease containing two FADD-like domains

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2897%2901282-9

Involvement of MACH, a novel MORT1/FADD-interacting protease, in Fas/APO-1- and TNF receptor-induced cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2897%2901282-9

FLICE, a novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death--inducing signaling complex

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2897%2901282-9

Fas involvement in Ca(2+)-independent T cell-mediated cytotoxicity

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2897%2901282-9

Regulation of the Fas apoptotic cell death pathway by Abl.

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2897%2901282-9

Anthracyclines and their C-13 alcohol metabolites: growth inhibition and DNA damage following incubation with human tumor cells in culture

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2897%2901282-9

Detection of tumor necrosis factor induced nuclear damage with p-phenylenediamine

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2897%2901282-9

Early redistribution of plasma membrane phosphatidylserine is a general feature of apoptosis regardless of the initiating stimulus: inhibition by overexpression of Bcl-2 and Abl

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2897%2901282-9

Increased intracellular cyclic AMP inhibits inositol phospholipid hydrolysis induced by perturbation of the T cell receptor/CD3 complex but not by G-protein stimulation. Association with protein kinase A-mediated phosphorylation of phospholipase C-g

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2897%2901282-9

Defective expression of hematopoietic cell protein tyrosine phosphatase (HCP) in lymphoid cells blocks Fas-mediated apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2897%2901282-9

Role of oxidative damage and IL-1 beta-converting enzyme-like proteases in Fas-based cytotoxicity exerted by effector T cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2897%2901282-9

Benzyloxycarbonyl-Val-Ala-Asp (OMe) fluoromethylketone (Z-VAD.FMK) inhibits apoptosis by blocking the processing of CPP32.

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2897%2901282-9

The Ced-3/interleukin 1beta converting enzyme-like homolog Mch6 and the lamin-cleaving enzyme Mch2alpha are substrates for the apoptotic mediator CPP32

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2897%2901282-9

Studies of the lamin proteinase reveal multiple parallel biochemical pathways during apoptotic execution

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2897%2901282-9

Lamin proteolysis facilitates nuclear events during apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2897%2901282-9

Involvement of the CD95 (APO–1/Fas) receptor/ligand system in drug–induced apoptosis in leukemia cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2897%2901282-9

T cell receptor-induced Fas ligand expression in cytotoxic T lymphocyte clones is blocked by protein tyrosine kinase inhibitors and cyclosporin A

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2897%2901282-9

Immunotargeting of liposomes containing lipophilic antitumor prodrugs

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2897%2901282-9

Aspartate-based inhibitor of interleukin-1 beta-converting enzyme prevents antitumor agent-induced apoptosis in human myeloid leukemia U937 cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2897%2901282-9

Daunorubicin-induced apoptosis: triggering of ceramide generation through sphingomyelin hydrolysis

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2897%2901282-9

10.1016/S0014-5793(97)01282-9

7 January 2021

Identifiers

DOI

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26 September 2017

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