Camptothecin causes cell cycle perturbations within T-lymphoblastoid cells followed by dose dependent induction of apoptosis (Q41076134)

26 September 2017

title

Camptothecin causes cell cycle perturbations within T-lymphoblastoid cells followed by dose dependent induction of apoptosis (English)

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26 September 2017

Johnson N

1

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26 September 2017

Ng TT

series ordinal

2

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26 September 2017

Parkin JM

series ordinal

3

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26 September 2017

publication date

1 October 1997

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26 September 2017

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26 September 2017

volume

21

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26 September 2017

issue

10

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26 September 2017

page(s)

961-972

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Fas and FasL in the homeostatic regulation of immune responses

26 September 2017

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Induction of differentiation of human and mouse myeloid leukemia cells by camptothecin

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Dose-dependent induction of apoptosis in human tumour cell lines by widely diverging stimuli.

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Apoptosis of S-phase HL-60 cells induced by DNA topoisomerase inhibitors: detection of DNA strand breaks by flow cytometry using the in situ nick translation assay

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A rapid and simple method for measuring thymocyte apoptosis by propidium iodide staining and flow cytometry

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Cell death: the significance of apoptosis

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Distinct patterns of cell cycle disturbance elicited by compounds interfering with DNA topoisomerase I and II activity

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Effect of camptothecin on mitogenic stimulation of human lymphocytes: involvement of DNA topoisomerase I in cell transition from G0 to G1 phase of the cell cycle and in DNA replication.

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Specific phosphorylation of SR proteins by mammalian DNA topoisomerase I.

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A death in the life of p53

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p53 is required for radiation-induced apoptosis in mouse thymocytes

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Thymocyte apoptosis induced by p53-dependent and independent pathways.

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p53 and Sp1 interact and cooperate in the tumor necrosis factor-induced transcriptional activation of the HIV-1 long terminal repeat

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Identifiers

DOI

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26 September 2017

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