Hypoxia-induced modulation of endothelial cell properties: regulation of barrier function and expression of interleukin-6. (Q41347387)

29 September 2017

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title

Hypoxia-induced modulation of endothelial cell properties: regulation of barrier function and expression of interleukin-6. (English)

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29 September 2017

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S F Yan

1

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29 September 2017

S Ogawa

series ordinal

2

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29 September 2017

D M Stern

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3

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29 September 2017

D J Pinsky

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4

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29 September 2017

language of work or name

English

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publication date

1 February 1997

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29 September 2017

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29 September 2017

51

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29 September 2017

419-425

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29 September 2017

2

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https://scigraph.springernature.com/pub.10.1038/ki.1997.56

29 September 2017

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cites work

Hypoxia modulates the barrier and coagulant function of cultured bovine endothelium. Increased monolayer permeability and induction of procoagulant properties

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.56

7 January 2021

Macrovascular and microvascular endothelium during long-term hypoxia: alterations in cell growth, monolayer permeability, and cell surface coagulant properties

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https://api.crossref.org/works/10.1038%2FKI.1997.56

7 January 2021

Hypoxia induces glucose transporter expression in endothelial cells

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.56

7 January 2021

Hypoxia and mitochondrial inhibitors regulate expression of glucose transporter-1 via distinct Cis-acting sequences

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.56

7 January 2021

Rapid increase in blood-brain barrier permeability during severe hypoxia and metabolic inhibition.

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.56

7 January 2021

Permeability characteristics of cultured endothelial cell monolayers

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.56

7 January 2021

Tumor necrosis factor/cachectin increases permeability of endothelial cell monolayers by a mechanism involving regulatory G proteins

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.56

7 January 2021

Hypoxia-induced increased permeability of endothelial monolayers occurs through lowering of cellular cAMP levels

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https://api.crossref.org/works/10.1038%2FKI.1997.56

7 January 2021

Diminished polymorphonuclear leukocyte adherence. Function dependent on release of cyclic AMP by endothelial cells after stimulation of beta-receptors by epinephrine

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.56

7 January 2021

Restoration of the cAMP second messenger pathway enhances cardiac preservation for transplantation in a heterotopic rat model

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.56

7 January 2021

Cardiac preservation is enhanced in a heterotopic rat transplant model by supplementing the nitric oxide pathway

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.56

7 January 2021

The nitric oxide/cyclic GMP pathway in organ transplantation: critical role in successful lung preservation

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.56

7 January 2021

Hypoxia-mediated induction of endothelial cell interleukin-1 alpha. An autocrine mechanism promoting expression of leukocyte adhesion molecules on the vessel surface

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.56

7 January 2021

Hypoxic induction of interleukin-8 gene expression in human endothelial cells

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.56

7 January 2021

Ischemia-induced interleukin-8 release after human heart transplantation. A potential role for endothelial cells

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.56

7 January 2021

Induction of interleukin 6 (IL-6) by hypoxia in vascular cells. Central role of the binding site for nuclear factor-IL-6.

1 reference

https://api.crossref.org/works/10.1038%2FKI.1997.56

7 January 2021

Identifiers

DOI

10.1038/KI.1997.56

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29 September 2017

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