Does an acidic pH explain why low density lipoprotein is oxidised in atherosclerotic lesions? (Q41432893)

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scientific article published on March 1997

Language	Label	Description	Also known as
English	Does an acidic pH explain why low density lipoprotein is oxidised in atherosclerotic lesions?	scientific article published on March 1997

Statements

scholarly article

1 reference

Europe PubMed Central

30 September 2017

1 reference

Europe PubMed Central

Does an acidic pH explain why low density lipoprotein is oxidised in atherosclerotic lesions? (English)

1 reference

Europe PubMed Central

30 September 2017

atherosclerosis

1 reference

based on heuristic

inferred from title

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based on heuristic

inferred from title

author name string

Leake DS

1

1 reference

Europe PubMed Central

30 September 2017

publication date

1 March 1997

1 reference

Europe PubMed Central

30 September 2017

Atherosclerosis

1 reference

Europe PubMed Central

30 September 2017

129

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Europe PubMed Central

30 September 2017

2

1 reference

Europe PubMed Central

30 September 2017

149-157

1 reference

Europe PubMed Central

30 September 2017

The role of oxidized lipoproteins in atherogenesis

1 reference

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Enhanced macrophage degradation of biologically modified low density lipoprotein.

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Macrophage oxidation of low density lipoprotein generates a modified form recognized by the scavenger receptor

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The oxidative modification of low-density lipoproteins by macrophages

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The oxidative modification of low density lipoprotein by human lymphocytes

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Iron and copper promote modification of low density lipoprotein by human arterial smooth muscle cells in culture

1 reference

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Acidic pH enables caeruloplasmin to catalyse the modification of low-density lipoprotein

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Macrophages require both iron and copper to oxidize low-density lipoprotein in Hanks' balanced salt solution

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Oxidation of low-density lipoprotein by thiol compounds leads to its recognition by the acetyl LDL receptor

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Presence of foam cells containing oxidised low density lipoprotein in the synovial membrane from patients with rheumatoid arthritis

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Oxidized low-density lipoprotein in experimental focal glomerulosclerosis

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Human suction blister interstitial fluid prevents metal ion-dependent oxidation of low density lipoprotein by macrophages and in cell-free systems

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Distribution of plasma proteins across the human aortic wall Barrier functions of endothelium and internal elastic lamina

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Acidic pH increases the oxidation of LDL by macrophages

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Lipoprotein oxidation and lipoprotein-induced cytotoxicity

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The action of defined oxygen-centred free radicals on human low-density lipoprotein

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Effect of pH on low-density lipoprotein oxidation by O2-./HO2. free radicals produced by gamma radiolysis

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Myeloperoxidase, a catalyst for lipoprotein oxidation, is expressed in human atherosclerotic lesions

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Cholesterol chlorohydrin synthesis by the myeloperoxidase-hydrogen peroxide-chloride system: potential markers for lipoproteins oxidatively damaged by phagocytes

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Presence of hypochlorite-modified proteins in human atherosclerotic lesions

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A comparison of the structure and properties of serum transferrin from 17 animal species

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The mechanism of iron release from transferrin. Slow-proton-transfer-induced loss of nitrilotriacetatoiron(III) complex in acidic media

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Iron released from transferrin at acidic pH can catalyse the oxidation of low density lipoprotein

1 reference

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Stimulation of lipid peroxidation and hydroxyl-radical generation by the contents of human atherosclerotic lesions

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Prooxidant iron and copper, with ferroxidase and xanthine oxidase activities in human atherosclerotic material.

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Transition metal ions within human atherosclerotic lesions can catalyse the oxidation of low density lipoprotein by macrophages

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High stored iron levels are associated with excess risk of myocardial infarction in eastern Finnish men.

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Low iron-binding capacity as a risk factor for myocardial infarction

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Stored iron levels and myocardial infarction at young age

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Iron status and coronary heart disease: negative findings from the NHANES I epidemiologic follow-up study

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Body iron stores and the risk of coronary heart disease

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The modulation of ferryl myoglobin formation and its oxidative effects on low density lipoproteins by nitric oxide

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The interaction between ruptured erythrocytes and low‐density lipoproteins

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The state of the copper sites in human ceruloplasmin

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Lipoprotein oxidation and induction of ferroxidase activity in stored human extracellular fluids

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Intact human ceruloplasmin oxidatively modifies low density lipoprotein

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Role of endogenous ceruloplasmin in low density lipoprotein oxidation by human U937 monocytic cells

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Low density lipoprotein oxidation by stimulated neutrophils and ferritin

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Peroxynitrite releases copper from caeruloplasmin: implications for atherosclerosis.

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Age effect on brain pH during ischemia/reperfusion and pH influence on peroxidation.

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based on heuristic

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Identifiers

10.1016/S0021-9150(96)06035-2

1 reference

Europe PubMed Central

30 September 2017

1 reference

Europe PubMed Central

30 September 2017

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