Increased hepcidin levels in high-altitude pulmonary edema (Q41590698)

4 October 2017

title

Increased hepcidin levels in high-altitude pulmonary edema (English)

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4 October 2017

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2

Peter Bärtsch

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4

Marco Maggiorini

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7

Martina U Muckenthaler

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8

Heimo Mairbäurl

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3

Christoph Dehnert

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5

Günter Weiss

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Sandro Altamura

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Igor Theurl

series ordinal

6

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language of work or name

English

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publication date

18 December 2014

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Journal of Applied Physiology

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published in

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volume

118

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issue

3

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page(s)

292-298

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Prevention of high-altitude pulmonary edema by nifedipine

4 October 2017

cites work

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No evidence for interstitial lung oedema by extensive pulmonary function testing at 4,559 m.

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Hepcidin expression in human airway epithelial cells is regulated by interferon-γ.

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High-altitude pulmonary edema is initially caused by an increase in capillary pressure.

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Unchanged in vivo P50 at high altitude despite decreased erythrocyte age and elevated 2,3-diphosphoglycerate

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Hepcidin, a putative mediator of anemia of inflammation, is a type II acute-phase protein

21 January 2018

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Modulation of hepcidin production during hypoxia-induced erythropoiesis in humans in vivo: data from the HIGHCARE project

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21 January 2018

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The increase in pulmonary arterial pressure caused by hypoxia depends on iron status

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Hypoxia induced downregulation of hepcidin is mediated by platelet derived growth factor BB.

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Pathogenesis of high-altitude pulmonary edema: inflammation is not an etiologic factor

21 January 2018

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High levels of GDF15 in thalassemia suppress expression of the iron regulatory protein hepcidin

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Identification of TWSG1 as a second novel erythroid regulator of hepcidin expression in murine and human cells

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Identification of individuals susceptible to high-altitude pulmonary oedema at low altitude

21 January 2018

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Regulation of hepcidin expression at high altitude

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10.1152/JAPPLPHYSIOL.00940.2014

21 January 2018

Identifiers

DOI

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4 October 2017

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