Detection of hypoxic cells with the 2-nitroimidazole, EF5, correlates with early redox changes in rat brain after perinatal hypoxia-ischemia. (Q41671014)

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scientific article published on January 1999

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English	Detection of hypoxic cells with the 2-nitroimidazole, EF5, correlates with early redox changes in rat brain after perinatal hypoxia-ischemia.	scientific article published on January 1999

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scholarly article

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8 December 2019

Detection of hypoxic cells with the 2-nitroimidazole, EF5, correlates with early redox changes in rat brain after perinatal hypoxia-ischemia (English)

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8 December 2019

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Sydney M. Evans

2

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8 December 2019

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M Bergeron

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8 December 2019

F R Sharp

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8 December 2019

C J Koch

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8 December 2019

E M Lord

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8 December 2019

D M Ferriero

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8 December 2019

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1 January 1999

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8 December 2019

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8 December 2019

89

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8 December 2019

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8 December 2019

1357-1366

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Ischemic delayed neuronal death. A mitochondrial hypothesis.

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Identification of hypoxia in cells and tissues of epigastric 9L rat glioma using EF5 [2-(2-nitro-1H-imidazol-1-yl)-N-(2,2,3,3,3-pentafluoropropyl) acetamide].

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DNA fragmentation indicative of apoptosis following unilateral cerebral hypoxia-ischemia in the neonatal rat

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The role of specific reductases in the intracellular activation and binding of 2-nitroimidazoles

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Oxygen dependence of cellular uptake of EF5 [2-(2-nitro-1H-imidazol-1-yl)-N-(2,2,3,3,3-pentafluoropropyl)a cet amide] : analysis of drug adducts by fluorescent antibodies vs bound radioactivity

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The influence of immaturity on hypoxic‐ischemic brain damage in the rat

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Regional cerebral blood flow during hypoxia-ischemia in the immature rat: comparison of iodoantipyrine and iodoamphetamine as radioactive tracers.

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Altered mitochondrial respiration in selectively vulnerable brain subregions following transient forebrain ischemia in the rat.

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Neuropathology of remote hypoxic-ischemic damage in the immature rat

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Columnar alterations of NADH fluorescence during hypoxia-ischemia in immature rat brain.

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Identifiers

10.1016/S0306-4522(98)00377-7

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