Inhibition of serine palmitoyl transferase I reduces cardiac ceramide levels and increases glycolysis rates following diet-induced insulin resistance. (Q41813859)

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scientific article published on 22 May 2012

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English	Inhibition of serine palmitoyl transferase I reduces cardiac ceramide levels and increases glycolysis rates following diet-induced insulin resistance.	scientific article published on 22 May 2012

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2 February 2020

Inhibition of serine palmitoyl transferase I reduces cardiac ceramide levels and increases glycolysis rates following diet-induced insulin resistance (English)

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2 February 2020

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insulin resistance

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2 February 2020

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John R Ussher

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2 February 2020

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Clifford D L Folmes

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Natasha Fillmore

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2 February 2020

Jagdip S Jaswal

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Virgilio J Cadete

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2 February 2020

Donna L Beker

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2 February 2020

Victoria H Lam

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Liyan Zhang

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Gary D Lopaschuk

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22 May 2012

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2 February 2020

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2 February 2020

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2 February 2020

e37703

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2 February 2020

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Creative Commons Attribution 4.0 International

22 May 2012

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Diabetic downregulation of Nrf2 activity via ERK contributes to oxidative stress-induced insulin resistance in cardiac cells in vitro and in vivo

1 reference

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Inhibition of de novo ceramide synthesis reverses diet-induced insulin resistance and enhances whole-body oxygen consumption

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Myocardium of type 2 diabetic and obese patients is characterized by alterations in sphingolipid metabolic enzymes but not by accumulation of ceramide.

1 reference

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Insulin-stimulated cardiac glucose oxidation is increased in high-fat diet-induced obese mice lacking malonyl CoA decarboxylase

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6 June 2018

Central role of ceramide biosynthesis in body weight regulation, energy metabolism, and the metabolic syndrome

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6 June 2018

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Ceramide is a cardiotoxin in lipotoxic cardiomyopathy

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Ceramides in insulin resistance and lipotoxicity

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The obesity epidemic, metabolic syndrome and future prevention strategies

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Impairment of glucose metabolism in hearts from rats treated with endotoxin

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6 June 2018

High levels of fatty acids delay the recovery of intracellular pH and cardiac efficiency in post-ischemic hearts by inhibiting glucose oxidation

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A novel mouse model of lipotoxic cardiomyopathy

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1 reference

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1 reference

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inferred from PubMed ID database lookup

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1 reference

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10.1371/JOURNAL.PONE.0037703

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2 February 2020

2012PLoSO...737703U

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2 February 2020

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2 February 2020

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