Overexpression of granulocyte-macrophage colony-stimulating factor induces pulmonary granulation tissue formation and fibrosis by induction of transforming growth factor-beta 1 and myofibroblast accumulation (Q41938862)

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scientific article published on January 1997

Language	Label	Description	Also known as
English	Overexpression of granulocyte-macrophage colony-stimulating factor induces pulmonary granulation tissue formation and fibrosis by induction of transforming growth factor-beta 1 and myofibroblast accumulation	scientific article published on January 1997

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scholarly article

1 reference

Europe PubMed Central

14 October 2017

Overexpression of granulocyte-macrophage colony-stimulating factor induces pulmonary granulation tissue formation and fibrosis by induction of transforming growth factor-beta 1 and myofibroblast accumulation (English)

1 reference

Europe PubMed Central

14 October 2017

1 reference

based on heuristic

inferred from title

author name string

Xing Z

1

1 reference

Europe PubMed Central

14 October 2017

Tremblay GM

2

1 reference

Europe PubMed Central

14 October 2017

Sime PJ

3

1 reference

Europe PubMed Central

14 October 2017

Gauldie J

4

1 reference

Europe PubMed Central

14 October 2017

language of work or name

0 references

publication date

1 January 1997

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Europe PubMed Central

14 October 2017

The American Journal of Pathology

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Europe PubMed Central

14 October 2017

150

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Europe PubMed Central

14 October 2017

1

1 reference

Europe PubMed Central

14 October 2017

59-66

1 reference

Europe PubMed Central

14 October 2017

Gene transfer for cytokine functional studies in the lung: the multifunctional role of GM-CSF in pulmonary inflammation.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=1858539

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Transfer of granulocyte-macrophage colony-stimulating factor gene to rat lung induces eosinophilia, monocytosis, and fibrotic reactions

1 reference

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Cytoskeletal protein modulation in pulmonary alveolar myofibroblasts during idiopathic pulmonary fibrosis. Possible role of transforming growth factor beta and tumor necrosis factor alpha.

1 reference

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Transforming growth factor-beta 1 induces alpha-smooth muscle actin expression in granulation tissue myofibroblasts and in quiescent and growing cultured fibroblasts

1 reference

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Glomerulosclerosis induced by in vivo transfection of transforming growth factor-beta or platelet-derived growth factor gene into the rat kidney

1 reference

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Cytokines and pulmonary fibrosis

1 reference

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Polymorphonuclear leukocytes as a significant source of tumor necrosis factor-alpha in endotoxin-challenged lung tissue

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Cytokine expression by neutrophils and macrophages in vivo: endotoxin induces tumor necrosis factor-alpha, macrophage inflammatory protein-2, interleukin-1 beta, and interleukin-6 but not RANTES or transforming growth factor-beta 1 mRNA expression i

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BCG-induced granuloma formation in murine tissues

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Regulation of alveolar macrophage transforming growth factor-beta secretion by corticosteroids in bleomycin-induced pulmonary inflammation in the rat.

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Cytokines involved in pulmonary fibrosis.

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Factors influencing myofibroblast differentiation during wound healing and fibrosis.

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Increased central nervous system production of extracellular matrix components and development of hydrocephalus in transgenic mice overexpressing transforming growth factor-beta 1

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Cellular localization of transforming growth factor-beta expression in bleomycin-induced pulmonary fibrosis

1 reference

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Expression of a tumor necrosis factor-alpha transgene in murine lung causes lymphocytic and fibrosing alveolitis. A mouse model of progressive pulmonary fibrosis

1 reference

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Myofibroblasts and their role in lung collagen gene expression during pulmonary fibrosis. A combined immunohistochemical and in situ hybridization study

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Rat alveolar myofibroblasts acquire alpha-smooth muscle actin expression during bleomycin-induced pulmonary fibrosis

1 reference

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Pathobiology of pulmonary fibrosis

1 reference

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Alpha-smooth muscle actin is transiently expressed by myofibroblasts during experimental wound healing.

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Fibrogenic cytokines: the role of immune mediators in the development of scar tissue

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Role of transforming growth factor-beta in repair and fibrosis

1 reference

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Transforming growth factor beta 1 is present at sites of extracellular matrix gene expression in human pulmonary fibrosis

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Tumor necrosis factor. Characterization at the molecular, cellular and in vivo level

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5 June 2018

Lung cytokine production in bleomycin-induced pulmonary fibrosis

1 reference

https://pubmed.ncbi.nlm.nih.gov/9006322

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Increased production and immunohistochemical localization of transforming growth factor-beta in idiopathic pulmonary fibrosis.

1 reference

https://pubmed.ncbi.nlm.nih.gov/9006322

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

GM-CSF-induced granulation tissue formation: relationships between macrophage and myofibroblast accumulation

1 reference

https://pubmed.ncbi.nlm.nih.gov/9006322

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Distribution of transforming growth factor-beta 1, fibronectin, and smooth muscle actin in asbestos-induced pulmonary fibrosis in rats

1 reference

https://pubmed.ncbi.nlm.nih.gov/9006322

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

TGF-beta 1, but not TGF-beta 2 or TGF-beta 3, is differentially present in epithelial cells of advanced pulmonary fibrosis: an immunohistochemical study

1 reference

https://pubmed.ncbi.nlm.nih.gov/9006322

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Granulocyte- and granulocyte– macrophage-colony stimulating factors induce human endothelial cells to migrate and proliferate

1 reference

https://pubmed.ncbi.nlm.nih.gov/9006322

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Idiopathic pulmonary fibrosis. A paradigm for lung injury and repair

1 reference

https://pubmed.ncbi.nlm.nih.gov/9006322

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Locally applied GM-CSF induces the accumulation of alpha-smooth muscle actin containing myofibroblasts

1 reference

https://pubmed.ncbi.nlm.nih.gov/9006322

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

1 reference

Europe PubMed Central

14 October 2017

1 reference

Europe PubMed Central

14 October 2017

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