N terminus is key to the dominant negative suppression of Ca(V)2 calcium channels: implications for episodic ataxia type 2. (Q41960046)

14 October 2017

N terminus is key to the dominant negative suppression of Ca(V)2 calcium channels: implications for episodic ataxia type 2. (English)

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14 October 2017

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Manuela Nieto-Rostro

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14 October 2017

Karen M Page

1

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Fay Heblich

2

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Wojciech Margas

3

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Wendy S Pratt

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Kanchan Chaggar

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14 October 2017

Kieran Sandhu

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14 October 2017

Anthony Davies

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14 October 2017

language of work or name

English

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publication date

10 November 2009

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Journal of Biological Chemistry

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285

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2

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835-844

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describes a project that uses

14 October 2017

ImageQuant

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11 June 2022

https://www.ebi.ac.uk/europepmc/webservices/rest/PMC2801285/fullTextXML

based on heuristic

inferred from PubMed Central ID database lookup

cites work

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Dominant-negative effects of episodic ataxia type 2 mutations involve disruption of membrane trafficking of human P/Q-type Ca2+ channels.

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Functional consequences of P/Q-type Ca2+ channel Cav2.1 missense mutations associated with episodic ataxia type 2 and progressive ataxia

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Molecular basis for K(ATP) assembly: transmembrane interactions mediate association of a K+ channel with an ABC transporter

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High prevalence of CACNA1A truncations and broader clinical spectrum in episodic ataxia type 2

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Familial hemiplegic migraine and episodic ataxia type-2 are caused by mutations in the Ca2+ channel gene CACNL1A4

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Voltage-dependent calcium channel beta-subunits in combination with alpha 1 subunits, have a GTPase activating effect to promote the hydrolysis of GTP by G alpha o in rat frontal cortex

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Identification of residues in the N terminus of alpha1B critical for inhibition of the voltage-dependent calcium channel by Gbeta gamma.

18 August 2018

1 reference

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Identification of the amino terminus of neuronal Ca2+ channel alpha1 subunits alpha1B and alpha1E as an essential determinant of G-protein modulation.

18 August 2018

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Nonsense-mediated mRNA decay

18 August 2018

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PubMed

https://pubmed.ncbi.nlm.nih.gov/19903821

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Dominant-negative synthesis suppression of voltage-gated calcium channel Cav2.2 induced by truncated constructs

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/19903821

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1074/JBC.M109.065045

1 reference

14 October 2017

1 reference

14 October 2017

PubMed publication ID

19903821

1 reference