A slowly ADP-ribosylated pertussis-toxin-sensitive GTP-binding regulatory protein is required for vasopressin-stimulated Ca2+ inflow in hepatocytes (Q41975626)

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scientific article published on April 1994

Language	Label	Description	Also known as
English	A slowly ADP-ribosylated pertussis-toxin-sensitive GTP-binding regulatory protein is required for vasopressin-stimulated Ca2+ inflow in hepatocytes	scientific article published on April 1994

Statements

scholarly article

1 reference

Europe PubMed Central

14 October 2017

A slowly ADP-ribosylated pertussis-toxin-sensitive GTP-binding regulatory protein is required for vasopressin-stimulated Ca2+ inflow in hepatocytes (English)

1 reference

Europe PubMed Central

14 October 2017

0 references

author name string

Berven LA

1

1 reference

Europe PubMed Central

14 October 2017

Hughes BP

2

1 reference

Europe PubMed Central

14 October 2017

Barritt GJ

3

1 reference

Europe PubMed Central

14 October 2017

language of work or name

0 references

publication date

1 April 1994

1 reference

Europe PubMed Central

14 October 2017

Biochemical Journal

1 reference

Europe PubMed Central

14 October 2017

299 ( Pt 2)

1 reference

Europe PubMed Central

14 October 2017

399-407

1 reference

Europe PubMed Central

14 October 2017

Endoplasmic reticulum-through-Golgi transport assay based on O-glycosylation of native glycophorin in permeabilized erythroleukemia cells: role for Gi3.

1 reference

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Coupling of the expressed alpha 1B-adrenergic receptor to the phospholipase C pathway in Xenopus oocytes. The role of Go.

1 reference

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The involvement of ankyrin in the regulation of inositol 1,4,5-trisphosphate receptor-mediated internal Ca2+ release from Ca2+ storage vesicles in mouse T-lymphoma cells

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Activation of phospholipase C isozymes by G protein beta gamma subunits.

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Flash photolysis of caged inositol 1,4,5-trisphosphate activates plasma membrane calcium current in human T cells

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Noradrenaline, vasopressin and angiotensin increase Ca2+ influx by opening a common pool of Ca2+ channels in isolated rat liver cells

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High-yield preparation of isolated rat liver parenchymal cells: a biochemical and fine structural study

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Stimulation of inositol trisphosphate formation in hepatocytes by vasopressin, adrenaline and angiotensin II and its relationship to changes in cytosolic free Ca2+.

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Evidence that a pertussis-toxin-sensitive substrate is involved in the stimulation by epidermal growth factor and vasopressin of plasma-membrane Ca2+ inflow in hepatocytes

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The stimulation by sodium fluoride of plasma-membrane Ca2+ inflow in isolated hepatocytes. Evidence that a GTP-binding regulatory protein is involved in the hormonal stimulation of Ca2+ inflow

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G proteins: transducers of receptor-generated signals

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Effect of islet-activating pertussis toxin on the binding characteristics of Ca2+-mobilizing hormones and on agonist activation of phosphorylase in hepatocytes

1 reference

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Characterization of cytosolic calcium oscillations induced by phenylephrine and vasopressin in single fura-2-loaded hepatocytes

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Effects of heparin on inositol 1,4,5-trisphosphate and guanosine 5'-O-(3-thio triphosphate) induced calcium release in cultured smooth muscle cells from rabbit trachea

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Evidence that guanosine 5'-[gamma-thio]triphosphate stimulates plasma membrane Ca2+ inflow when introduced into hepatocytes

1 reference

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G protein subunit, alpha i-3, activates a pertussis toxin-sensitive Na+ channel from the epithelial cell line, A6.

1 reference

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Primary structure and functional expression of the inositol 1,4,5-trisphosphate-binding protein P400.

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Coupling of exogenous receptors to phospholipase C in Xenopus oocytes through pertussis toxin-sensitive and -insensitive pathways. Cross-talk through heterotrimeric G-proteins

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Ca2+ oscillations induced by hormonal stimulation of individual fura-2-loaded hepatocytes.

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Cyclic GMP-activated conductance of retinal photoreceptor cells

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'Quantal' Ca2+ release and the control of Ca2+ entry by inositol phosphates--a possible mechanism

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Receptor-operated calcium influx in rat hepatocytes. Identification and characterization using manganese

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Liver inositol, 1,4,5-trisphosphate-binding sites are the Ca2(+)-mobilizing receptors

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The liver cell plasma membrane Ca2+ inflow systems exhibit a broad specificity for divalent metal ions

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Cyclic GMP mediates the agonist-stimulated increase in plasma membrane calcium entry in the pancreatic acinar cell

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Capacitative calcium entry revisited

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Inhibition of Ca2+ inflow causes an abrupt cessation of growth-factor-induced repetitive free Ca2+ transients in single NIH-3T3 cells

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Inhibitory GTP-binding regulatory protein Gi3 can couple angiotensin II receptors to inhibition of adenylyl cyclase in hepatocytes.

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Subcellular distribution of the calcium-storing inositol 1,4,5-trisphosphate-sensitive organelle in rat liver. Possible linkage to the plasma membrane through the actin microfilaments

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Pertussis toxin-sensitive Gi protein involvement in epidermal growth factor-induced activation of phospholipase C-gamma in rat hepatocytes

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The epidermal growth factor receptor is coupled to a pertussis toxin-sensitive guanine nucleotide regulatory protein in rat hepatocytes

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Characteristics of membrane currents evoked by photoreleased inositol trisphosphate in Xenopus oocytes.

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Actions of vasopressin and the Ca(2+)-ATPase inhibitor, thapsigargin, on Ca2+ signaling in hepatocytes

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Subunits beta gamma of heterotrimeric G protein activate beta 2 isoform of phospholipase C

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10.1042/BJ2990399

1 reference

Europe PubMed Central

14 October 2017

1 reference

Europe PubMed Central

14 October 2017

1 reference

Europe PubMed Central

14 October 2017

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