Mitochondrial ATP-sensitive potassium channels enhance angiotensin-induced oxidative damage and dopaminergic neuron degeneration. Relevance for aging-associated susceptibility to Parkinson's disease (Q42136286)

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scientific article published on 29 June 2011

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English	Mitochondrial ATP-sensitive potassium channels enhance angiotensin-induced oxidative damage and dopaminergic neuron degeneration. Relevance for aging-associated susceptibility to Parkinson's disease	scientific article published on 29 June 2011

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scholarly article

1 reference

Europe PubMed Central

PMC publication ID

17 October 2017

Mitochondrial ATP-sensitive potassium channels enhance angiotensin-induced oxidative damage and dopaminergic neuron degeneration. Relevance for aging-associated susceptibility to Parkinson's disease (English)

1 reference

Europe PubMed Central

PMC publication ID

17 October 2017

Parkinson's disease

1 reference

based on heuristic

inferred from title

1 reference

based on heuristic

inferred from title

dopaminergic neuron

1 reference

based on heuristic

inferred from title

Jannette Rodriguez-Pallares

object named as

Jannette Rodriguez-Pallares

1

0 references

author name string

Juan Andres Parga

2

1 reference

Europe PubMed Central

PMC publication ID

17 October 2017

Belen Joglar

3

1 reference

Europe PubMed Central

PMC publication ID

17 October 2017

Maria Jose Guerra

4

1 reference

Europe PubMed Central

PMC publication ID

17 October 2017

Jose Luis Labandeira-Garcia

5

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Europe PubMed Central

PMC publication ID

17 October 2017

publication date

29 June 2011

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Europe PubMed Central

PMC publication ID

17 October 2017

number of pages

18

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Europe PubMed Central

PMC publication ID

17 October 2017

34

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Europe PubMed Central

PMC publication ID

17 October 2017

4

1 reference

Europe PubMed Central

PMC publication ID

17 October 2017

863-880

1 reference

Europe PubMed Central

PMC publication ID

17 October 2017

describes a project that uses

1 reference

Europe PubMed Central

11 June 2022

https://www.ebi.ac.uk/europepmc/webservices/rest/PMC3682060/fullTextXML

based on heuristic

inferred from PubMed Central ID database lookup

Angiotensin II revisited: new roles in inflammation, immunology and aging

1 reference

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Missing pieces in the Parkinson's disease puzzle

1 reference

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Therapeutic targeting of mitochondrial superoxide in hypertension

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Redox signaling (cross-talk) from and to mitochondria involves mitochondrial pores and reactive oxygen species

1 reference

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Potassium channels in brain mitochondria

1 reference

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Live longer sans the AT1A receptor

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Cross-talk between mitochondria and NADPH oxidase: effects of mild mitochondrial dysfunction on angiotensin II-mediated increase in Nox isoform expression and activity in vascular smooth muscle cells

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Mitochondrial potassium channels and uncoupling proteins in synaptic plasticity and neuronal cell death

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Changes in somatodendritic but not terminal dopamine regulation in aged rhesus monkeys

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Role of the renin-angiotensin system in vascular diseases: expanding the field.

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MitoTracker labeling in primary neuronal and astrocytic cultures: influence of mitochondrial membrane potential and oxidants.

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Bioenergetic consequences of opening the ATP-sensitive K(+) channel of heart mitochondria

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NADPH oxidase: an update

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Cardioprotective effect of diazoxide and its interaction with mitochondrial ATP-sensitive K+ channels. Possible mechanism of cardioprotection.

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Neuronal vulnerability in Parkinson's disease.

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Oxidation reactions in Parkinson's disease.

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29 May 2018

The oxidant stress hypothesis in Parkinson's disease: evidence supporting it

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Aging-related changes in the nigral angiotensin system enhances proinflammatory and pro-oxidative markers and 6-OHDA-induced dopaminergic degeneration

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Opening mitoKATP increases superoxide generation from complex I of the electron transport chain

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Identifiers

10.1007/S11357-011-9284-7

1 reference

Europe PubMed Central

PMC publication ID

17 October 2017

PMC publication ID

1 reference

Europe PubMed Central

PMC publication ID

17 October 2017

PubMed publication ID

1 reference

Europe PubMed Central

PMC publication ID

17 October 2017

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