Creatine kinase-deficient hearts exhibit increased susceptibility to ischemia-reperfusion injury and impaired calcium homeostasis (Q42460676)

1 November 2017

Creatine kinase-deficient hearts exhibit increased susceptibility to ischemia-reperfusion injury and impaired calcium homeostasis (English)

1 reference

1 November 2017

1 reference

7

Stefan Neubauer

1 reference

1 November 2017

1

Matthias Spindler

1 reference

1 November 2017

3

Hinrik Strömer

1 reference

1 November 2017

Klaus Meyer

2

1 reference

1 November 2017

Andrea Leupold

series ordinal

4

1 reference

1 November 2017

Ernest Boehm

series ordinal

5

1 reference

1 November 2017

Helga Wagner

series ordinal

6

1 reference

1 November 2017

22 April 2004

1 reference

American Journal of Physiology Heart and Circulatory Physiology

1 November 2017

1 reference

1 November 2017

287

1 reference

1 November 2017

3

1 reference

1 November 2017

H1039-45

1 reference

Glycolysis supports calcium uptake by the sarcoplasmic reticulum in skinned ventricular fibres of mice deficient in mitochondrial and cytosolic creatine kinase.

1 November 2017

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01016.2003

Role of creatine kinase in cardiac excitation-contraction coupling: studies in creatine kinase-deficient mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01016.2003

Simultaneous measurement of intracellular calcium and ventricular function in the phospholamban-deficient mouse heart

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01016.2003

Ischemic contracture of the myocardium: mechanisms and prevention

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01016.2003

Energetic crosstalk between organelles: architectural integration of energy production and utilization

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01016.2003

Excitation-contraction coupling in postischemic myocardium. Does failure of activator Ca2+ transients underlie stunning?

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01016.2003

Pathophysiology and pathogenesis of stunned myocardium. Depressed Ca2+ activation of contraction as a consequence of reperfusion-induced cellular calcium overload in ferret hearts

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01016.2003

High calcium and dobutamine positive inotropy in the perfused mouse heart: myofilament calcium responsiveness, energetic economy, and effects of protein kinase C inhibition

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01016.2003

Kinetic, thermodynamic, and developmental consequences of deleting creatine kinase isoenzymes from the heart. Reaction kinetics of the creatine kinase isoenzymes in the intact heart.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01016.2003

Diastolic dysfunction and altered energetics in the alphaMHC403/+ mouse model of familial hypertrophic cardiomyopathy

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01016.2003

Correlation between cytosolic free calcium, contracture, ATP, and irreversible ischemic injury in perfused rat heart

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01016.2003

Role of MgADP in the development of diastolic dysfunction in the intact beating rat heart

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01016.2003

Intracellular compartmentation, structure and function of creatine kinase isoenzymes in tissues with high and fluctuating energy demands: the 'phosphocreatine circuit' for cellular energy homeostasis

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.01016.2003

10.1152/AJPHEART.01016.2003

21 January 2018

Identifiers

DOI

1 reference

1 November 2017

1 reference