Lysophosphatidylcholine Increases Ca Current via Activation of Protein Kinase C in Rabbit Portal Vein Smooth Muscle Cells. (Q42467344)

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18 January 2020

title

Lysophosphatidylcholine Increases Ca Current via Activation of Protein Kinase C in Rabbit Portal Vein Smooth Muscle Cells (English)

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18 January 2020

2

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Duck Sun Ahn

6

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18 January 2020

author name string

Seungsoo Jung

1

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18 January 2020

Sungsik Han

3

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Youngwhan Kim

4

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18 January 2020

Taiksang Nam

5

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publication date

28 February 2008

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Korean Journal of Physiology and Pharmacology

18 January 2020

published in

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volume

12

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issue

1

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18 January 2020

page(s)

31-35

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Creative Commons Attribution-NonCommercial 3.0 Unported

18 January 2020

copyright license

start time

1 January 2008

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April 2022 Public Data File from Crossref

copyright status

copyrighted

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cites work

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Use of protein kinase C inhibitors results in rapid [Mg(2+)](i) mobilization in primary cultured rat aortic smooth muscle cells: are certain kinase C isoforms natural homeostatic regulators of cystolic free Mg(2+).

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PKC activity modulates availability and long openings of L-type Ca2+ channels in A7r5 cells.

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Regulation of 4-aminopyridine-sensitive, delayed rectifier K+ channels in vascular smooth muscle by phosphorylation

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Effects of oxidized low-density lipoprotein on vascular contraction and relaxation: clinical and pharmacological implications in atherosclerosis

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Recent insights pertaining to sarcolemmal phospholipid alterations underlying arrhythmogenesis in the ischemic heart.

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Staurosporine, K-252 and UCN-01: potent but nonspecific inhibitors of protein kinases

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The role of membrane biophysical properties in the regulation of protein kinase C activity

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Lysophosphatidylcholine decreases delayed rectifier K+ current in rabbit coronary smooth muscle cells

28 May 2018

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Exogenous lysophosphatidylcholine increases non-selective cation current in guinea-pig ventricular myocytes

16 August 2018

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Lysophosphatidylcholine inhibits endothelium-dependent hyperpolarization and N omega-nitro-L-arginine/indomethacin-resistant endothelium-dependent relaxation in the porcine coronary artery.

16 August 2018

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Lysophosphatidylcholine causes cGMP-dependent verapamil-sensitive Ca2+ influx in vascular smooth muscle cells.

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Alpha 2-adrenoceptors activate dihydropyridine-sensitive calcium channels via Gi-proteins and protein kinase C in rat portal vein myocytes

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12 December 2020

Lysophosphatidylcholine promotes P-selectin expression in platelets and endothelial cells. Possible involvement of protein kinase C activation and its inhibition by nitric oxide donors

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12 December 2020

Altered contractile and ion channel function in rabbit portal vein with dietary atherosclerosis

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12 December 2020

Lysophosphatidylcholine causes Ca2+ influx, enhanced DNA synthesis and cytotoxicity in cultured vascular smooth muscle cells

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12 December 2020

Protein kinase C stimulates Ca2+ current in pregnant rat myometrial cells

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12 December 2020

Lysophosphatidylcholine in oxidized low-density lipoprotein increases endothelial susceptibility to polymorphonuclear leukocyte-induced endothelial dysfunction in porcine coronary arteries. Role of protein kinase C

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12 December 2020

Selective enhancement of 5-hydroxytryptamine-induced contraction of porcine coronary artery by oxidized low-density lipoprotein

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12 December 2020

Mildly oxidized LDL evokes a sustained Ca(2+)-dependent retraction of vascular smooth muscle cells

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12 December 2020

Lysophosphatidylcholine triggers intracellular calcium release and activation of non-selective cation channels in renal arterial smooth muscle cells

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12 December 2020

Oxidized low density lipoproteins potentiate vasoconstrictions to various agonists by direct interaction with vascular smooth muscle

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12 December 2020

LPC in oxidized LDL elicits vasocontraction and inhibits endothelium- dependent relaxation

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12 December 2020

Angiotensin II activation of protein kinase C decreases delayed rectifier K+ current in rabbit vascular myocytes

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12 December 2020

Lysophosphatidylcholine modulates cardiac I(Na) via multiple protein kinase pathways

1 reference

12 December 2020

10.4196/KJPP.2008.12.1.31

Identifiers

DOI

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18 January 2020

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18 January 2020

PubMed publication ID

20157391

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