An altered redox balance mediates the hypersensitivity of Cockayne syndrome primary fibroblasts to oxidative stress. (Q42503209)

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25 January 2020

title

An altered redox balance mediates the hypersensitivity of Cockayne syndrome primary fibroblasts to oxidative stress (English)

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1

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Egidio Iorio

3

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author name string

Tiziana Lemma

2

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Sara Giovannini

4

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Bruno Vaz

5

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Ivano Iavarone

6

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Angelo Calcagnile

7

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Laura Narciso

8

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Paolo Degan

9

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Franca Podo

10

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Vera Roginskya

11

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Bratislav M Janjic

12

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Bennett Van Houten

13

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Miria Stefanini

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Eugenia Dogliotti

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Mariarosaria D'Errico

16

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publication date

5 April 2012

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volume

11

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issue

3

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page(s)

520-529

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Cockayne syndrome group B protein promotes mitochondrial DNA stability by supporting the DNA repair association with the mitochondrial membrane

25 January 2020

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Defective repair of 5-hydroxy-2'-deoxycytidine in Cockayne syndrome cells and its complementation by Escherichia coli formamidopyrimidine DNA glycosylase and endonuclease III.

21 January 2018

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Cockayne syndrome B protein (CSB): linking p53, HIF-1 and p300 to robustness, lifespan, cancer and cell fate decisions.

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Proteins of nucleotide and base excision repair pathways interact in mitochondria to protect from loss of subcutaneous fat, a hallmark of aging

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8-Oxoguanine DNA glycosylase (Ogg1) causes a transcriptional inactivation of damaged DNA in the absence of functional Cockayne syndrome B (Csb) protein.

21 January 2018

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Xeroderma pigmentosum, trichothiodystrophy and Cockayne syndrome: a complex genotype-phenotype relationship

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Cockayne syndrome group B protein stimulates repair of formamidopyrimidines by NEIL1 DNA glycosylase

21 January 2018

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Evaluation of the probes 2′,7′-dichlorofluorescin diacetate, luminol, and lucigenin as indicators of reactive species formation

21 January 2018

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Cockayne syndrome group B protein (CSB) plays a general role in chromatin maintenance and remodeling

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Primary fibroblasts of Cockayne syndrome patients are defective in cellular repair of 8-hydroxyguanine and 8-hydroxyadenine resulting from oxidative stress

21 January 2018

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Impaired genome maintenance suppresses the growth hormone--insulin-like growth factor 1 axis in mice with Cockayne syndrome

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Cockayne syndrome B protein stimulates apurinic endonuclease 1 activity and protects against agents that introduce base excision repair intermediates

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Mitochondrial dysfunction and oxidative stress mediate the physiological impairment induced by the disruption of autophagy

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21 January 2018

Identifiers

DOI

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25 January 2020

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